Mechanisms in which preconditioning and mitochondrial ATP-sensitive 'K POT. +' channels protect against damage promoted by ischemia/reperfusion (2007)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Subjects: ISQUEMIA MIOCÁRDICA; MITOCÔNDRIAS; BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Source:
- Título: Abstracts
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular
-
ABNT
FACUNDO, Heberty Di Tarso Fernandes e PAULA, Juliana Gabriela de e KOWALTOWSKI, Alicia Juliana. Mechanisms in which preconditioning and mitochondrial ATP-sensitive 'K POT. +' channels protect against damage promoted by ischemia/reperfusion. 2007, Anais.. São Paulo: SBBq, 2007. . Acesso em: 20 jan. 2026. -
APA
Facundo, H. D. T. F., Paula, J. G. de, & Kowaltowski, A. J. (2007). Mechanisms in which preconditioning and mitochondrial ATP-sensitive 'K POT. +' channels protect against damage promoted by ischemia/reperfusion. In Abstracts. São Paulo: SBBq. -
NLM
Facundo HDTF, Paula JG de, Kowaltowski AJ. Mechanisms in which preconditioning and mitochondrial ATP-sensitive 'K POT. +' channels protect against damage promoted by ischemia/reperfusion. Abstracts. 2007 ;[citado 2026 jan. 20 ] -
Vancouver
Facundo HDTF, Paula JG de, Kowaltowski AJ. Mechanisms in which preconditioning and mitochondrial ATP-sensitive 'K POT. +' channels protect against damage promoted by ischemia/reperfusion. Abstracts. 2007 ;[citado 2026 jan. 20 ] - Higher respiratory activity decreases mitochondrial reactive oxygen release and increases life span in Saccharomyces cerevisiae
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- Redox properties of ATP-sensitive 'K POT. +' channels in brain mitochondria
- Mitochondrial ATP-sensitive 'K POT. +' channels
- Respiration, oxidative stress and chronological life span in S. cerevisiae
- ATP-sensitive 'K POT +'channels in brain mitochondria are redox-sensitive and regulate reactive oxygen release
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Phosphate uptake increases mitochondrial H2O2 release
- How mitochondrial complex II inhibition promotes neuronal damage
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