Synthetic prostacyclin analogs differentially regulate macrophage function via distinct analog-receptor binding specificities (2007)
- Authors:
- Aronoff, David M. - University of Michigan (UM)
- Peres, Camila M.
- Serezani, Carlos H. - University of Michigan (UM)
- Ballinger, Megan N. - University of Michigan (UM)
- Carstens, Jennifer K. - University of Michigan (UM)
- Coleman, Nicole - University of Michigan (UM)
- Moore, Bethany B. - University of Michigan (UM)
- Peebles, R. Stokes - Vanderbilt University (VU)
- Faccioli, Lúcia Helena
- Peters-Golden, Marc - University of Michigan (UM)
- Autor USP: FACCIOLI, LUCIA HELENA - FCFRP
- Unidade: FCFRP
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Immunology
- ISSN: 0022-1767
- Volume/Número/Paginação/Ano: v. 178, n. 3, p. 1628-1634, 2007
-
ABNT
ARONOFF, David M. et al. Synthetic prostacyclin analogs differentially regulate macrophage function via distinct analog-receptor binding specificities. Journal of Immunology, v. 178, n. 3, p. 1628-1634, 2007Tradução . . Acesso em: 19 set. 2024. -
APA
Aronoff, D. M., Peres, C. M., Serezani, C. H., Ballinger, M. N., Carstens, J. K., Coleman, N., et al. (2007). Synthetic prostacyclin analogs differentially regulate macrophage function via distinct analog-receptor binding specificities. Journal of Immunology, 178( 3), 1628-1634. -
NLM
Aronoff DM, Peres CM, Serezani CH, Ballinger MN, Carstens JK, Coleman N, Moore BB, Peebles RS, Faccioli LH, Peters-Golden M. Synthetic prostacyclin analogs differentially regulate macrophage function via distinct analog-receptor binding specificities. Journal of Immunology. 2007 ; 178( 3): 1628-1634.[citado 2024 set. 19 ] -
Vancouver
Aronoff DM, Peres CM, Serezani CH, Ballinger MN, Carstens JK, Coleman N, Moore BB, Peebles RS, Faccioli LH, Peters-Golden M. Synthetic prostacyclin analogs differentially regulate macrophage function via distinct analog-receptor binding specificities. Journal of Immunology. 2007 ; 178( 3): 1628-1634.[citado 2024 set. 19 ] - Eosinophil migration to lung in helminthic infection is not inhibited by glucocorticoids
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