Ishemic preconditioning enhances fatty acid-dependent mitochondrial uncoupling (2007)
- Authors:
- USP affiliated authors: MASCIO, PAOLO DI - IQ ; KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Subjects: ÁCIDOS GRAXOS; MITOCÔNDRIAS; BIOQUÍMICA; ISQUEMIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Abstracts
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular
-
ABNT
CARREIRA, Raquel S. et al. Ishemic preconditioning enhances fatty acid-dependent mitochondrial uncoupling. 2007, Anais.. São Paulo: SBBq, 2007. . Acesso em: 20 abr. 2024. -
APA
Carreira, R. S., Miyamoto, S., Di Mascio, P., Monteiro, P., Gonçalves, L. M., Providência, L. A., & Kowaltowski, A. J. (2007). Ishemic preconditioning enhances fatty acid-dependent mitochondrial uncoupling. In Abstracts. São Paulo: SBBq. -
NLM
Carreira RS, Miyamoto S, Di Mascio P, Monteiro P, Gonçalves LM, Providência LA, Kowaltowski AJ. Ishemic preconditioning enhances fatty acid-dependent mitochondrial uncoupling. Abstracts. 2007 ;[citado 2024 abr. 20 ] -
Vancouver
Carreira RS, Miyamoto S, Di Mascio P, Monteiro P, Gonçalves LM, Providência LA, Kowaltowski AJ. Ishemic preconditioning enhances fatty acid-dependent mitochondrial uncoupling. Abstracts. 2007 ;[citado 2024 abr. 20 ] - Modification of cytochrome C and increased mitocondrial oxygen comsuption induced by Trans,trans-2,4-decadienal
- trans, trans-2,4-decadienal induces mitochondrial dysfunction and oxidative stress
- Ischemic preconditioning enhances fatty acid-dependent mitochondrial uncoupling
- Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
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