Modulatory effect of arginase in NO production by macrophage activated by Bordetella pertussis (2007)
- Authors:
- Autor USP: ABRAHAMSOHN, ISES DE ALMEIDA - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Imunologia
- Publisher place: Rio de Janeiro
- Date published: 2007
- Source:
- Título: Abstracts
- Conference titles: International Congress of Immunology
-
ABNT
ROSETTI, A. S. et al. Modulatory effect of arginase in NO production by macrophage activated by Bordetella pertussis. 2007, Anais.. Rio de Janeiro: Sociedade Brasileira de Imunologia, 2007. . Acesso em: 02 out. 2024. -
APA
Rosetti, A. S., Neto, J. A., Carvalho, V. D., Abrahamsohn, I. de A., & Borges, M. M. (2007). Modulatory effect of arginase in NO production by macrophage activated by Bordetella pertussis. In Abstracts. Rio de Janeiro: Sociedade Brasileira de Imunologia. -
NLM
Rosetti AS, Neto JA, Carvalho VD, Abrahamsohn I de A, Borges MM. Modulatory effect of arginase in NO production by macrophage activated by Bordetella pertussis. Abstracts. 2007 ;[citado 2024 out. 02 ] -
Vancouver
Rosetti AS, Neto JA, Carvalho VD, Abrahamsohn I de A, Borges MM. Modulatory effect of arginase in NO production by macrophage activated by Bordetella pertussis. Abstracts. 2007 ;[citado 2024 out. 02 ] - Endogenously produced IL-10 and prostaglandins regulate peripheral blood mononuclear cells proliferation to Trypanosoma cruzi antigens
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- Egulation of Trypanosoma cruzi infection by type interferons
- Il-12 regulation of ifn-gama synthesis in the acute phase of trypanosoma cruzi infection
- IL-12 regulation of IFN-'gama' synthesis in the acute phase of Trypanosoma cruzi infection
- Prostaglandins mediate suppression of lymphoproliferation and cytokine synthesis during the early phase of Trypanosoma cruzi infection
- Il-12 enhances proliferation of peripheral blood mononuclear cells from chagasic patients to trypanosoma cruzi antigen
- Il-10 and ifn-gamma production by spleen cells in trypanosoma cruzi infected mice
- Bordetella pertussis and innate immune response: inhibition of NO production by murine BMDMO through TGF-ß arginase and TLR4
- Immature macrophages derived from mouse bone marrow produce large amounts of IL-12p40 after LPS stimulation
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