Increased influx of activated T cells to the lungs and high TNF-'alfa' secretion are associated with less severe Paracoccidioidomycosis developed by iNOS KO mice (2007)
- Authors:
- USP affiliated author: CALICH, VERA LUCIA GARCIA - ICB
- School: ICB
- Subject: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Imunologia
- Place of publication: Rio de Janeiro
- Date published: 2007
- Source:
- Título do periódico: Abstracts
- Conference title: International Congress of Immunology
-
ABNT
BERNARDINO, S.; CALICH, Vera Lúcia Garcia. Increased influx of activated T cells to the lungs and high TNF-'alfa' secretion are associated with less severe Paracoccidioidomycosis developed by iNOS KO mice. Anais.. Rio de Janeiro: Sociedade Brasileira de Imunologia, 2007. -
APA
Bernardino, S., & Calich, V. L. G. (2007). Increased influx of activated T cells to the lungs and high TNF-'alfa' secretion are associated with less severe Paracoccidioidomycosis developed by iNOS KO mice. In Abstracts. Rio de Janeiro: Sociedade Brasileira de Imunologia. -
NLM
Bernardino S, Calich VLG. Increased influx of activated T cells to the lungs and high TNF-'alfa' secretion are associated with less severe Paracoccidioidomycosis developed by iNOS KO mice. Abstracts. 2007 ; -
Vancouver
Bernardino S, Calich VLG. Increased influx of activated T cells to the lungs and high TNF-'alfa' secretion are associated with less severe Paracoccidioidomycosis developed by iNOS KO mice. Abstracts. 2007 ; - Role of IL-12 in the innate immunity of athymic and euthymic balb/c mice with paracoccidioidomycosis: effect of local and systemic il-12 neutralization
- Pulmonary paracoccidioidomycosis (PCM) in IL-10 knock-out (KO) mice
- Genetic deficiency of IL-10 determines a less severe paracoccidioidomycosis associated with increased IL-12 secretion and enhanced T cell activation
- Genetic deficiency of CD28 costimulatory molecule results in more severe Paracoccicioidomycosis (PCM) associated with decreased antibodies and cytokines production
- Paracoccidioides brasiliensis-TLR-4 interaction induces macrophage activation but results in more severe paracoccidioimycosis
- CD28 costimulatory molecule deficiency results in more severe Paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Suceptible and resistant mice to p. brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth
- Susceptible and resistant mice to P. Brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth
- Mannose receptors play a different role in the activation of macrophages from resistant and susceptible mice to Paracoccidioides brasiliensis infection
- CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis
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