LmYPT overexpression abolishes HTBF-mediated terbinafine resistance in Leishmania major (2005)
- Authors:
- Autor USP: TOSI, LUIZ RICARDO ORSINI - FMRP
- Unidade: FMRP
- Assunto: LEISHMANIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Proceedings
- Conference titles: Meeting of The Brazilian Society of Protozoology
-
ABNT
PINTO, Marcelo R. e TOSI, Luiz Ricardo Orsini. LmYPT overexpression abolishes HTBF-mediated terbinafine resistance in Leishmania major. 2005, Anais.. Caxambu: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2005. . Acesso em: 27 jul. 2024. -
APA
Pinto, M. R., & Tosi, L. R. O. (2005). LmYPT overexpression abolishes HTBF-mediated terbinafine resistance in Leishmania major. In Proceedings. Caxambu: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Pinto MR, Tosi LRO. LmYPT overexpression abolishes HTBF-mediated terbinafine resistance in Leishmania major. Proceedings. 2005 ;[citado 2024 jul. 27 ] -
Vancouver
Pinto MR, Tosi LRO. LmYPT overexpression abolishes HTBF-mediated terbinafine resistance in Leishmania major. Proceedings. 2005 ;[citado 2024 jul. 27 ] - The 9-1-1 complex is involved in cell cycle checkpoint and gene amplification modulation in the protozoan Leishmania
- Isolamento e clonagem da RAB/GTPase YPT em Leishmania SPP
- The YIP1 protein of Leishmania major and its role in terbinafine resistance
- Characterization of differences in the expression of epissomal genes in leishmania major and leishmania braziliensis
- The role of chromosome context in site-directed disruption of subtelometric targets of Leishimania major
- YIP1 of Leishmania major: characterization and possible role in terbinafine resistance
- Leishmania major nuclear protein LMHUS1 protects DNA from damage
- A transposon toolkit for gene transfer and mutagenesis in protozoan parasites
- LmHus1 is required in the DNA damage response in Leishmania major and forms a complex with an unusual Rad9 homologue
- DiCre-based conditional knockout of LMHUS1 reveals functional compartmentalization of the 9-1-1 complex in the DNA damage response of Leishmania major
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