Neutrophil migration failure and cardiovascular dysfunction in severe sepsis induced by CLP in mice: peroxynitrite role (2005)
- Authors:
- USP affiliated authors: ROSSI, MARCOS ANTONIO - FMRP ; FERREIRA, SERGIO HENRIQUE - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidade: FMRP
- Subjects: FARMACOLOGIA; INFLAMAÇÃO; DOR
- Language: Inglês
- Imprenta:
- Publisher: FMRP-USP
- Publisher place: Ribeirão Preto
- Date published: 2005
- Source:
- Título do periódico: Abstracts
- Conference titles: Jornada Farmacológica
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ABNT
TORRES-DUEÑAS, Diego et al. Neutrophil migration failure and cardiovascular dysfunction in severe sepsis induced by CLP in mice: peroxynitrite role. 2005, Anais.. Ribeirão Preto: FMRP-USP, 2005. . Acesso em: 29 set. 2024. -
APA
Torres-Dueñas, D., Celis, M. R., Farias-Filho, J. C., Spiller, F., Freitas, A., Dal Secco, D., et al. (2005). Neutrophil migration failure and cardiovascular dysfunction in severe sepsis induced by CLP in mice: peroxynitrite role. In Abstracts. Ribeirão Preto: FMRP-USP. -
NLM
Torres-Dueñas D, Celis MR, Farias-Filho JC, Spiller F, Freitas A, Dal Secco D, Rossi MA, Ferreira SH, Cunha F de Q. Neutrophil migration failure and cardiovascular dysfunction in severe sepsis induced by CLP in mice: peroxynitrite role. Abstracts. 2005 ;[citado 2024 set. 29 ] -
Vancouver
Torres-Dueñas D, Celis MR, Farias-Filho JC, Spiller F, Freitas A, Dal Secco D, Rossi MA, Ferreira SH, Cunha F de Q. Neutrophil migration failure and cardiovascular dysfunction in severe sepsis induced by CLP in mice: peroxynitrite role. Abstracts. 2005 ;[citado 2024 set. 29 ] - Hydrogen sulfide improves neutrophil migration and survival in sepsis via 'K POT.+' ATP channel activation
- Peroxynitrite mediates the failure of neutrophil migration in severe polymicrobial sepsis in mice
- Hydrogen sulfide augments neutrophil migration through enhancement of adhesion molecule expression and prevention of CXCR2 internalization: role of ATP-Sensitive potassium channels
- Nitric oxide inhibits neutrophil migration by a mechanism dependent on ICAM-1: role of soluble guanylate cyclase
- Reduction of ICAM-1 expression by carbon monoxide via soluble guanylate cyclase activation accounts for modulation of neutrophil migration
- Cardiac changes in experimental sepsis
- Cardiac changes in experimental sepsis
- Reduction of gap junction proteins and intercalated disc structural remodeling in the hearts of mice submitted to sepsis
- Sepsis and the heart
- Reduction of gap and adherens junction proteins and intercalated disc structural remodeling in the hearts of mice submitted to severe cecal ligation and puncture sepsis
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