Endothelial cells contribute to isoprenaline and terbutaline-induced vascular relaxation in normotensive 2K-SO and renal hypertensive 2K-1C rat aorta (2005)
- Authors:
- USP affiliated author: BENDHACK, LUSIANE MARIA - FCFRP
- School: FCFRP
- Subject: FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Autonomic Neuroscience : Basic and Clinical
- ISSN: 1566-0702
- Volume/Número/Paginação/Ano: v. 119, special issue, p. 127, 2005
- Conference title: Meeting of International Society for Autonomic Neuroscience (ISAN
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ABNT
BENDHACK, Lusiane Maria e ANJOS, Mário Neto Filho. Endothelial cells contribute to isoprenaline and terbutaline-induced vascular relaxation in normotensive 2K-SO and renal hypertensive 2K-1C rat aorta. Autonomic Neuroscience : Basic and Clinical. Amsterdam: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 26 jun. 2022. , 2005 -
APA
Bendhack, L. M., & Anjos, M. N. F. (2005). Endothelial cells contribute to isoprenaline and terbutaline-induced vascular relaxation in normotensive 2K-SO and renal hypertensive 2K-1C rat aorta. Autonomic Neuroscience : Basic and Clinical. Amsterdam: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Bendhack LM, Anjos MNF. Endothelial cells contribute to isoprenaline and terbutaline-induced vascular relaxation in normotensive 2K-SO and renal hypertensive 2K-1C rat aorta. Autonomic Neuroscience : Basic and Clinical. 2005 ; 119 127.[citado 2022 jun. 26 ] -
Vancouver
Bendhack LM, Anjos MNF. Endothelial cells contribute to isoprenaline and terbutaline-induced vascular relaxation in normotensive 2K-SO and renal hypertensive 2K-1C rat aorta. Autonomic Neuroscience : Basic and Clinical. 2005 ; 119 127.[citado 2022 jun. 26 ] - Ouabain induces endothelial factors release in normotensive rats aorta
- A Ouabaína potencializa o efeito de altas concentrações de KCL na aorta de ratos hipertensos renais(2R-IC) com endotélio
- cGMP reduces 'CA POT.2+' influx in normotensive(2K) and in renal hypertensive(2K-1C) aortas
- Vascular reactivity to relaxing agents is not altered in rat aortas 2 hours after induction of sepsis
- New nitric oxide donnor reduces arterial pressure in renal hypertensive rats
- Vasodilatation induced by atrial natriuretic peptide(ANP) and C-type natriuretic peptide(CNP) in isolated aorta from rats
- Mecanismos celulares envolvidos na vasodilatação induzida pelo peptídeo natriurético atrial (ANP) e peptídeo natriurético tipo-C (CNP) em aorta isolada de ratos
- Comparison of the action mechanisms of sodium nitroprusside and [Ru(terpy)(bdq)NO+]3+ in normotensive and hypertensive rats (SHR)
- Various types of potassium channels are activated by the new nitric oxide (NO) donor [Ru(TERPY)(BDQ)NO+]3 + (TERPY) in mesenteric resistance arteries of renal hypertensive rats
- Non-selective inhibition of endothelial cyclooxygenase reduces the contractile response induced by phenylephrine in aorta from normotensive rats, but not in renal hypertensive rat aorta
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