Non-azotemic glomerulonephritic proteinuiric patients may have low turnover bone disease and their sera inhibidor osteoblastic proliferation in culture (2002)
- Authors:
- USP affiliated authors: BARROS, RUI TOLEDO - FM ; WORONIK, VIKTORIA - FM ; JORGETTI, VANDA - FM
- Unidade: FM
- Subjects: PROLIFERAÇÃO CELULAR; GLOMERUNEFRITE; OSTEOBLASTO
- Language: Inglês
- Imprenta:
- Publisher place: Washington
- Date published: 2002
- Source:
- Título: Journal of the American Society of Nephrology
- Volume/Número/Paginação/Ano: v. 13, p. 576A, res. SU-P0530
- Conference titles: ASN Renal Week
-
ABNT
DIAS, Cristiane B. et al. Non-azotemic glomerulonephritic proteinuiric patients may have low turnover bone disease and their sera inhibidor osteoblastic proliferation in culture. Journal of the American Society of Nephrology. Washington: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 28 dez. 2025. , 2002 -
APA
Dias, C. B., Gueiros, J. E. B., Reis, L. M. dos, Graciolli, F. J., Caparbo, V. F., Pereira, R. C., et al. (2002). Non-azotemic glomerulonephritic proteinuiric patients may have low turnover bone disease and their sera inhibidor osteoblastic proliferation in culture. Journal of the American Society of Nephrology. Washington: Faculdade de Medicina, Universidade de São Paulo. -
NLM
Dias CB, Gueiros JEB, Reis LM dos, Graciolli FJ, Caparbo VF, Pereira RC, Moyses RMA, Barros RT, Woronik V, Jorgetti V. Non-azotemic glomerulonephritic proteinuiric patients may have low turnover bone disease and their sera inhibidor osteoblastic proliferation in culture. Journal of the American Society of Nephrology. 2002 ; 13 576A.[citado 2025 dez. 28 ] -
Vancouver
Dias CB, Gueiros JEB, Reis LM dos, Graciolli FJ, Caparbo VF, Pereira RC, Moyses RMA, Barros RT, Woronik V, Jorgetti V. Non-azotemic glomerulonephritic proteinuiric patients may have low turnover bone disease and their sera inhibidor osteoblastic proliferation in culture. Journal of the American Society of Nephrology. 2002 ; 13 576A.[citado 2025 dez. 28 ] - ACE inibition reduces proteinuria, hematuria and renal expression of inflammatory mediators in humans lupus nephritis
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