Antagonism of glutamate receptors in the intermediate and caudal NTS of awake rats produced no changes in the hypertensive response to chemoreflex activation (2005)
- Authors:
- USP affiliated authors: MACHADO, BENEDITO HONORIO - FMRP ; BONAGAMBA, LENI GOMES HECK - FMRP
- Unidade: FMRP
- DOI: 10.1016/j.autneu.2004.10.004
- Assunto: HIPERTENSÃO (FISIOLOGIA)
- Language: Inglês
- Imprenta:
- Source:
- Título: Autonomic Neuroscience
- ISSN: 1566-0702
- Volume/Número/Paginação/Ano: v. 117, p. 25-32, 2005
- Este artigo NÃO possui versão em acesso aberto
-
Status: Nenhuma versão em acesso aberto identificada -
ABNT
MACHADO, Benedito Honório e BONAGAMBA, Leni Gomes Heck. Antagonism of glutamate receptors in the intermediate and caudal NTS of awake rats produced no changes in the hypertensive response to chemoreflex activation. Autonomic Neuroscience, v. 117, p. 25-32, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.autneu.2004.10.004. Acesso em: 11 mar. 2026. -
APA
Machado, B. H., & Bonagamba, L. G. H. (2005). Antagonism of glutamate receptors in the intermediate and caudal NTS of awake rats produced no changes in the hypertensive response to chemoreflex activation. Autonomic Neuroscience, 117, 25-32. doi:10.1016/j.autneu.2004.10.004 -
NLM
Machado BH, Bonagamba LGH. Antagonism of glutamate receptors in the intermediate and caudal NTS of awake rats produced no changes in the hypertensive response to chemoreflex activation [Internet]. Autonomic Neuroscience. 2005 ; 117 25-32.[citado 2026 mar. 11 ] Available from: https://doi.org/10.1016/j.autneu.2004.10.004 -
Vancouver
Machado BH, Bonagamba LGH. Antagonism of glutamate receptors in the intermediate and caudal NTS of awake rats produced no changes in the hypertensive response to chemoreflex activation [Internet]. Autonomic Neuroscience. 2005 ; 117 25-32.[citado 2026 mar. 11 ] Available from: https://doi.org/10.1016/j.autneu.2004.10.004 - Cardiovascular responses to chronic intermittent hypoxia in rats are not affected by previous exposure to hypoxia
- Microinjection of ionotropic and metabotropic selective agonists into the nts of conscious rats produces hypertension and bradycardia
- Minocyline attenuates the increase in arterial pressure of juvenile rats submitted to short-term sustained hypoxia
- Pressor response to chemoreflex activation in awake rats: role of vasopressin and adrenal medulla
- Hemodynamic and respiratory responses to microinjection of ATP into the intermediate and caudal NTS of awake rats
- Involvement of L-glutamate and ATP in the neurotransmission of the sympathoexcitatory component of the chemoreflex in the commissural nucleus tractus solitarii of awake rats and in the working heart-brainstem preparation
- Efeitos da microinjeção da substância P (SP) no núcleo do trato solitário (NTS) sobre o baro-e o quimiorreflexo (QR)
- Mechanisms of pressor and bradycardic responses to l-glutamate microinjected into the nts of conscious rats
- Resposta pressora a atividade do quimiorreflexo nao e bloqueada pela microinjecao de antagonistas ionotropicos ou metabotropicos no nts
- Microinjeção de l-glutamato (l-glu) no NTS comissural lateral de ratos acordados promove bradicardia que é mediada por receptores NMDA
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