Lectins KM+ and concanavalin a induce recruitment of mast cells from the bone marrow to the mesentery in rats (2004)
- Authors:
- USP affiliated authors: JAMUR, MARIA CELIA - FMRP ; BARREIRA, MARIA CRISTINA ROQUE ANTUNES - FMRP ; OLIVER, CONSTANCE - FMRP
- Unidade: FMRP
- Assunto: BIOLOGIA CELULAR
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Abstracts
- Conference titles: Congress of the Brazilian Society for the Cell Biology
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ABNT
BURANELLO, Patricia A. A. et al. Lectins KM+ and concanavalin a induce recruitment of mast cells from the bone marrow to the mesentery in rats. 2004, Anais.. Campinas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2004. . Acesso em: 18 set. 2024. -
APA
Buranello, P. A. A., Moulin, M. R. I., Roque-Barreira, M. C., Jamur, M. C., & Oliver, C. (2004). Lectins KM+ and concanavalin a induce recruitment of mast cells from the bone marrow to the mesentery in rats. In Abstracts. Campinas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Buranello PAA, Moulin MRI, Roque-Barreira MC, Jamur MC, Oliver C. Lectins KM+ and concanavalin a induce recruitment of mast cells from the bone marrow to the mesentery in rats. Abstracts. 2004 ;[citado 2024 set. 18 ] -
Vancouver
Buranello PAA, Moulin MRI, Roque-Barreira MC, Jamur MC, Oliver C. Lectins KM+ and concanavalin a induce recruitment of mast cells from the bone marrow to the mesentery in rats. Abstracts. 2004 ;[citado 2024 set. 18 ] - The expression of galectin-3 by inflammatory cells of mice experimentally infected with virulent Rhodococcus equi
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- Association between polymorphisms in genes that influences the immune response with cutaneous and Mucosal Leishmaniasis
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- Lectin MNCF induces polarization but not polymerization of F-actin filamentous in human neutrophils
- The lectin ArtinM binds to and is internalized by hematopoietic cell lines
- Lectin concanavalin a induce proliferation of mast cells of the bone marrow and the RBL-2H3
- The macrophage-derived lectin, MNCF, activates neutrophil migration through a pertussis toxin-sensitive pathway
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