Autonomously functioning thyroid nodules are not associated with BRAF mutations (2004)
- Authors:
- Autor USP: KIMURA, EDNA TERUKO - ICB
- Unidade: ICB
- Assunto: HISTOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: Blackwell Scientific Publications
- Publisher place: Oxford
- Date published: 2004
- Source:
- Título do periódico: Clinical Endocrinology
- ISSN: 0300-0664
- Volume/Número/Paginação/Ano: v. 60, p. 394-396, 2004
-
ABNT
KIMURA, Edna Teruko; VANVOOREN, Vanessa; SANDE, Jacqueline van; NIKIFOROV, Yuri E.; FAGIN, James A. Autonomously functioning thyroid nodules are not associated with BRAF mutations. Clinical Endocrinology[S.l: s.n.], 2004. -
APA
Kimura, E. T., Vanvooren, V., Sande, J. van, Nikiforov, Y. E., & Fagin, J. A. (2004). Autonomously functioning thyroid nodules are not associated with BRAF mutations. Clinical Endocrinology. Oxford: Blackwell Scientific Publications. -
NLM
Kimura ET, Vanvooren V, Sande J van, Nikiforov YE, Fagin JA. Autonomously functioning thyroid nodules are not associated with BRAF mutations. Clinical Endocrinology. 2004 ; 60 394-396. -
Vancouver
Kimura ET, Vanvooren V, Sande J van, Nikiforov YE, Fagin JA. Autonomously functioning thyroid nodules are not associated with BRAF mutations. Clinical Endocrinology. 2004 ; 60 394-396. - Oncongênese molecular das neoplasias da tiróide
- Notch activation is associated with tetraploidy and enhanced chromosomal instability in meningiomas
- TGFß, activina e sinalização SMAD em câncer de tiróide
- Estudo imunohistoquimico da proteina tgf beta3 em neoplasias benignas e malignas da glandula tiroide
- Expression of transforming growth factor beta (tgfbeta) 1 and 3 benign and malignant human thyroid glands
- Polyclonal and monoclonal thyroid nodules coexist within human multinodular goiters
- Enhanced expression of latent TGF binding protein (LTBP) in human thyroid tumors
- Acquired and naturally occurring resistance of thyroid follicular cells to the growth inhibitory action of transforming growth factor-beta1 (tgf-'BETA'1)
- Renovando, uma vez mais [Editorial]
- Galectina-3 está envolvida na via de sinalização WNT/'beta'- catenina e na transformação maligna do carcinoma papilífero de tiróide com rearranjo RET/PTC
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