Nongenetic male pseudohermaphroditism (2002)
- Authors:
- Autor USP: MENDONCA, BERENICE BILHARINHO DE - FM
- Unidade: FM
- Assunto: PSEUDO-HERMAFRODITISMO
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: New England Journal of Medicine
- Volume/Número/Paginação/Ano: v. 346, n. 8, p. 628-629, fev. 2002
-
ABNT
MENDONÇA, Berenice Bilharinho de; BILLERBECK, Ana Elisa C.; ZEGHER, Francis de. Nongenetic male pseudohermaphroditism. New England Journal of Medicine[S.l: s.n.], 2002. -
APA
Mendonça, B. B. de, Billerbeck, A. E. C., & Zegher, F. de. (2002). Nongenetic male pseudohermaphroditism. New England Journal of Medicine. Boston. -
NLM
Mendonça BB de, Billerbeck AEC, Zegher F de. Nongenetic male pseudohermaphroditism. New England Journal of Medicine. 2002 ; fe 2002( 8): 628-629. -
Vancouver
Mendonça BB de, Billerbeck AEC, Zegher F de. Nongenetic male pseudohermaphroditism. New England Journal of Medicine. 2002 ; fe 2002( 8): 628-629. - Molecular analysis of SRY gene in Brazilian 46,XX sex reversed patients: absense of SRY sequence in gonadal tissue
- Crescimento linear e estatura final em crianças com tumores adrenais predominantemente virilizantes
- Absense of inactivating mutations and deletions of DAX1 gene in SRY negative 46,XX sex-reversed patients
- Absense of duplication on DAX1 gene in patients with 46,XY sex reversal
- Heterogeneidade clínica e molecular do hipogonadismo hipogonadotrófico (HH): importância da avaliação do sistema olfatório no diagnóstico diferencial
- Number of CAG repeats in Exon 1 of androgen receptor gene is not related to gender identity in adult patients with male psudohermaphroditism and male transexualism
- Clinical, hormonal, behavioral, and genetic characteristics of androgen insensitibity syndrome in a brazilian cohort: five novel mutations in the androgen receptor gene
- Variable ACTH-stimulated 17-Hydroxyprogesterone values in 21-hydroxylase deficiency carriers are not related to the different CYP21 gene mutations
- Mutations in the SRY, DAX1, SF1 and WNT4 genes in brazilian sex-reversed patients
- Maternal isodisomy causing homozygosity for a dominant activating mutation of the luteinizing hormone receptor gene in a boy with familial male-limited precocius puberty
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