Androgen receptor CAG repeat plymorphism in prostate cancer from a Brazilian population (2003)
- Authors:
- Autor USP: NAGAI, MARIA APARECIDA - FM
- Unidade: FM
- DOI: 10.1016/s0361-090x(03)00106-5
- Subjects: NEOPLASIAS PROSTÁTICAS; RECEPTORES ANDROGÊNICOS; POLIMORFISMO
- Language: Inglês
- Imprenta:
- Source:
- Título: Cancer Detection and Prevention
- Volume/Número/Paginação/Ano: v. 27, p. 321-326, 2003
- Status:
- Nenhuma versão em acesso aberto identificada
-
ABNT
SANTOS, Mariana L. dos et al. Androgen receptor CAG repeat plymorphism in prostate cancer from a Brazilian population. Cancer Detection and Prevention, v. 27, p. 321-326, 2003Tradução . . Disponível em: https://doi.org/10.1016/s0361-090x(03)00106-5. Acesso em: 07 maio 2026. -
APA
Santos, M. L. dos, Sarkis, Á. S., Nishimoto, I. N., & Nagai, M. A. (2003). Androgen receptor CAG repeat plymorphism in prostate cancer from a Brazilian population. Cancer Detection and Prevention, 27, 321-326. doi:10.1016/s0361-090x(03)00106-5 -
NLM
Santos ML dos, Sarkis ÁS, Nishimoto IN, Nagai MA. Androgen receptor CAG repeat plymorphism in prostate cancer from a Brazilian population [Internet]. Cancer Detection and Prevention. 2003 ; 27 321-326.[citado 2026 maio 07 ] Available from: https://doi.org/10.1016/s0361-090x(03)00106-5 -
Vancouver
Santos ML dos, Sarkis ÁS, Nishimoto IN, Nagai MA. Androgen receptor CAG repeat plymorphism in prostate cancer from a Brazilian population [Internet]. Cancer Detection and Prevention. 2003 ; 27 321-326.[citado 2026 maio 07 ] Available from: https://doi.org/10.1016/s0361-090x(03)00106-5 - The transcripts of PHLDA1 and PAWR are modulated by '17 POT.2'-estradiol and IGF-1: extend analysis
- Identification of differentially expressed genes in the prostatic epithelium
- Opposing action of 17ß-estradiol on HNRPK and PAWR gene expression
- The CAG repeat polymorphism in the androgen receptor gene (AR) and its relationship to head and neck cancer
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- High expression of NDRG1 is associated with apocrine differention in breast tissue
- Gene expression profile and cancer-associated pathways linked to progesterone receptor isoform a (PRA) predominance in transgenic mouse mammary glands
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