Cytokine and cell recruitment induced by Strongyloides venezuelensis into the lung (2003)
- Authors:
- Autor USP: FACCIOLI, LUCIA HELENA - FCFRP
- Unidade: FCFRP
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Inflammation Research
- ISSN: 1023-3830
- Volume/Número/Paginação/Ano: v. 52, supl. 2, p. S 134, res. 26, 2003
- Conference titles: Word Congress on Inflammation
-
ABNT
MACHADO, Eleuza Rodrigues et al. Cytokine and cell recruitment induced by Strongyloides venezuelensis into the lung. Inflammation Research. Basel: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 23 jul. 2024. , 2003 -
APA
Machado, E. R., Ueta, M. T., Anibal, F. F., Oliveira, J. B. A., & Faccioli, L. H. (2003). Cytokine and cell recruitment induced by Strongyloides venezuelensis into the lung. Inflammation Research. Basel: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Machado ER, Ueta MT, Anibal FF, Oliveira JBA, Faccioli LH. Cytokine and cell recruitment induced by Strongyloides venezuelensis into the lung. Inflammation Research. 2003 ; 52 S 134.[citado 2024 jul. 23 ] -
Vancouver
Machado ER, Ueta MT, Anibal FF, Oliveira JBA, Faccioli LH. Cytokine and cell recruitment induced by Strongyloides venezuelensis into the lung. Inflammation Research. 2003 ; 52 S 134.[citado 2024 jul. 23 ] - Eosinophil migration to lung in helminthic infection is not inhibited by glucocorticoids
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- Estudo da reação inflamatória pulmonar induzida por Mycobacterium tuberculosis em cobaias vacinadas com DNA hsp65
- Role of glicocorticoids in the increase of eosinophils in blood and recruitment into peritoneal cavity and bronchoalveolar fluid of mice infected with S. venezuelensis
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- Inhibition of eosinophil recruitment in acute and chronic inflammation by ethanolic extract of Lafoensia pacari
- Role of leukotrienes in the cellular recruitment in mice infected with Strongyloides venezuelensis
- Prostaglandin E2-loaded microspheres as strategy to inhibit phagocytosis and modulate inflammatory mediators release
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