Ouabain induces endothelial factors release in normotensive rats aorta (2003)
- Authors:
- USP affiliated author: BENDHACK, LUSIANE MARIA - FCFRP
- School: FCFRP
- Subject: FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Place of publication: Ribeirão Preto
- Date published: 2003
- Conference title: Congresso da Sociedade Brasileira de Fisiologia
-
ABNT
MOLIN, Josiane Cristina et al. Ouabain induces endothelial factors release in normotensive rats aorta. 2003, Anais.. Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2003. . Acesso em: 26 jun. 2022. -
APA
Molin, J. C., Sguilla, F. dos S., Callera, G. E., & Bendhack, L. M. (2003). Ouabain induces endothelial factors release in normotensive rats aorta. In . Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Molin JC, Sguilla F dos S, Callera GE, Bendhack LM. Ouabain induces endothelial factors release in normotensive rats aorta. 2003 ;[citado 2022 jun. 26 ] -
Vancouver
Molin JC, Sguilla F dos S, Callera GE, Bendhack LM. Ouabain induces endothelial factors release in normotensive rats aorta. 2003 ;[citado 2022 jun. 26 ] - A Ouabaína potencializa o efeito de altas concentrações de KCL na aorta de ratos hipertensos renais(2R-IC) com endotélio
- cGMP reduces 'CA POT.2+' influx in normotensive(2K) and in renal hypertensive(2K-1C) aortas
- Endothelial cells contribute to isoprenaline and terbutaline-induced vascular relaxation in normotensive 2K-SO and renal hypertensive 2K-1C rat aorta
- Vascular reactivity to relaxing agents is not altered in rat aortas 2 hours after induction of sepsis
- New nitric oxide donnor reduces arterial pressure in renal hypertensive rats
- Vasodilatation induced by atrial natriuretic peptide(ANP) and C-type natriuretic peptide(CNP) in isolated aorta from rats
- Mecanismos celulares envolvidos na vasodilatação induzida pelo peptídeo natriurético atrial (ANP) e peptídeo natriurético tipo-C (CNP) em aorta isolada de ratos
- Comparison of the action mechanisms of sodium nitroprusside and [Ru(terpy)(bdq)NO+]3+ in normotensive and hypertensive rats (SHR)
- Various types of potassium channels are activated by the new nitric oxide (NO) donor [Ru(TERPY)(BDQ)NO+]3 + (TERPY) in mesenteric resistance arteries of renal hypertensive rats
- Non-selective inhibition of endothelial cyclooxygenase reduces the contractile response induced by phenylephrine in aorta from normotensive rats, but not in renal hypertensive rat aorta
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