Impaired relaxation to acetylcholine in 2K-1C hypertensive rat aortas involves changes in membrane hyperpolatization instead of an abnormal contribution of endothelial factors (2000)
- Authors:
- USP affiliated authors: VARANDA, WAMBERTO ANTONIO - FMRP ; BENDHACK, LUSIANE MARIA - FCFRP
- Unidades: FMRP; FCFRP
- DOI: 10.1016/s0306-3623(01)00075-1
- Assunto: FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: General Pharmacology
- ISSN: 0306-3623
- Volume/Número/Paginação/Ano: v. 34, p. 379-389, 2000
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
CALLERA, Gláucia Elena e VARANDA, Wamberto Antônio e BENDHACK, Lusiane Maria. Impaired relaxation to acetylcholine in 2K-1C hypertensive rat aortas involves changes in membrane hyperpolatization instead of an abnormal contribution of endothelial factors. General Pharmacology, v. 34, p. 379-389, 2000Tradução . . Disponível em: https://doi.org/10.1016/s0306-3623(01)00075-1. Acesso em: 09 jan. 2026. -
APA
Callera, G. E., Varanda, W. A., & Bendhack, L. M. (2000). Impaired relaxation to acetylcholine in 2K-1C hypertensive rat aortas involves changes in membrane hyperpolatization instead of an abnormal contribution of endothelial factors. General Pharmacology, 34, 379-389. doi:10.1016/s0306-3623(01)00075-1 -
NLM
Callera GE, Varanda WA, Bendhack LM. Impaired relaxation to acetylcholine in 2K-1C hypertensive rat aortas involves changes in membrane hyperpolatization instead of an abnormal contribution of endothelial factors [Internet]. General Pharmacology. 2000 ; 34 379-389.[citado 2026 jan. 09 ] Available from: https://doi.org/10.1016/s0306-3623(01)00075-1 -
Vancouver
Callera GE, Varanda WA, Bendhack LM. Impaired relaxation to acetylcholine in 2K-1C hypertensive rat aortas involves changes in membrane hyperpolatization instead of an abnormal contribution of endothelial factors [Internet]. General Pharmacology. 2000 ; 34 379-389.[citado 2026 jan. 09 ] Available from: https://doi.org/10.1016/s0306-3623(01)00075-1 - A contribuição dos canais para 'K POT.+' sensíveis ao 'Ca POT.2+' no relaxamento induzido com acetilcolina é menor em aortas de ratos hipertensos renais 2R-1C
- The function of voltage-gated 'Ca POT.2+' channels is increased in aortas from 2K-1C hypertensive rats
- Increased calcium influx in 2K-1C hypertensive rat aortas: role of calcium-activated channels
- Increased 'Ca POT. 2+' influx in 2K-1C hypertensive rat aortas: role of 'Ca POT. 2+'-activated 'K POT. +' channels
- 'Ca POT.2+' influx is increased in 2-kidney, 1-clip hypertensive rat aorta
- Effects of potassium channels inhibition in the relaxation to acetylcholine in aortas from 2k-1c hypertensive rats
- 'Ca pot. 2+' influx is increased in 2K-1C hypertensive rats aorta
- The function of voltage-gated C'A POT. 2+' channels is increased in aortas from 2K-lC hypertensive rats
- In vitro differentiation between oxytocin- and vasopressin-secreting magnocellular neurons requires more than one experimental criterion
- Receptores P2X em células de Leydig e ativação de canais para potássio ativados por cálcio
Informações sobre o DOI: 10.1016/s0306-3623(01)00075-1 (Fonte: oaDOI API)
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