Fungicidal activity in vivo of polymorphonuclear neutrophils on P. brasiliensis yeasts (2000)
- Authors:
- Autor USP: BURGER, EVA - ICB
- Unidade: ICB
- Assunto: IMMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: Brazilian Society of Immunology
- Publisher place: Florianópilis
- Date published: 2000
- Source:
- Título: Anais
- Conference titles: Meeting of the Brazilian Society of Immunology
-
ABNT
FERNANDES-BORDIGNON, G P e BURGER, Eva. Fungicidal activity in vivo of polymorphonuclear neutrophils on P. brasiliensis yeasts. 2000, Anais.. Florianópilis: Brazilian Society of Immunology, 2000. . Acesso em: 01 mar. 2026. -
APA
Fernandes-Bordignon, G. P., & Burger, E. (2000). Fungicidal activity in vivo of polymorphonuclear neutrophils on P. brasiliensis yeasts. In Anais. Florianópilis: Brazilian Society of Immunology. -
NLM
Fernandes-Bordignon GP, Burger E. Fungicidal activity in vivo of polymorphonuclear neutrophils on P. brasiliensis yeasts. Anais. 2000 ;[citado 2026 mar. 01 ] -
Vancouver
Fernandes-Bordignon GP, Burger E. Fungicidal activity in vivo of polymorphonuclear neutrophils on P. brasiliensis yeasts. Anais. 2000 ;[citado 2026 mar. 01 ] - Granulomatous lesions in experimental Paracoccidioides brasiliensis infection
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- Evaluation of paracoccidioidomycosis treatment with sulfamethoxazol-trimethoprin using immunological parameters in a murine model
- Concentration of IFN-'gama', IL-10, GM-CSF, KC, LTB-4, TNF-'alfa' and TGF-'beta' in air pouch of susceptible & resistant mice to "P. brasiliensis"
- Matrix metalloproteinases in murine experimental paracoccidioidomycosis: possible participation in the pattern of granulomatous lesions
- Differential cytokines expression pattern in granulomas of susceptible and resistant mice infected with a low virulent P. brasiliensis isolate
- Immune pattern reversion in acute murine paracoccidioidomycosis elicited by liposomal amphotericin B administration
- Alterations of granuloma architecture, cellularity and severity of the early infection in murine paracoccidioidomycosis by IFN-'alfa', celecoxib or lumiracoxib treatment
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