CD4(+) T cell responseto leishmania in non-infected individuals (2000)
- Autor:
- Autor USP: BARCINSKI, MARCELLO ANDRE - ICB
- Unidade: ICB
- Assunto: PARASITOLOGIA
- Language: Inglês
- Source:
- Título do periódico: Human Immunology
- ISSN: 0034-8910
- Volume/Número/Paginação/Ano: v.61,n.6,p. 238-45, 2000
-
ABNT
BARCINSKI, Marcelo André. CD4(+) T cell responseto leishmania in non-infected individuals. Human Immunology, v. 61, n. 6, p. 238-45, 2000Tradução . . Acesso em: 30 set. 2024. -
APA
Barcinski, M. A. (2000). CD4(+) T cell responseto leishmania in non-infected individuals. Human Immunology, 61(6), 238-45. -
NLM
Barcinski MA. CD4(+) T cell responseto leishmania in non-infected individuals. Human Immunology. 2000 ;61(6): 238-45.[citado 2024 set. 30 ] -
Vancouver
Barcinski MA. CD4(+) T cell responseto leishmania in non-infected individuals. Human Immunology. 2000 ;61(6): 238-45.[citado 2024 set. 30 ] - Identification of Leishmania spp metacyclic promastigotes by non-fluorescent flow cytometric parameters
- Phosphatidylserine expression on Leishmania metacyclic promastigotes
- Kinetics of exposure and distribution of phosphatidylserine on the surface of Leishmania (L) amazonensis amastigotes
- Granulocyte-macrophage colony-stimulating factor increases the infectivity of leishmania amazonensis by protecting promastigotes from heat-induced death
- Gm-csf protects promastigotes of leishmania (l) amazonensis from a calcium-modulated heat-induced death
- Protecao pelo gm-csf da morte de promastigotas de leishimania (l) amazonensis induzida pelo choque termico depende da fase do ciclo celular do parasito
- Strategies for molecular cloning and sequencing of programmed cell death putative genes in Leishmania (Leishmania) amazonensis promatigotes
- Bone-marrow stroma inhibits proliferation and apoptosis in leukemic cells through gap junction mediated cell communication
- Phosphatidylserine externalization and caspase-3-like activity in amastigotes of Leishmania SPP.: an apoptotic mimicry
- Normal hematopoiesis is maintained by activated bone marrow CD4+ T cells
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