Comparison between the effects of nimesulide and its reduced metabolite on mitochondria (2000)
- Authors:
- USP affiliated authors: SANTOS, ANTONIO CARDOZO DOS - FCFRP ; UYEMURA, SERGIO AKIRA - FCFRP ; CURTI, CARLOS - FCFRP
- School: FCFRP
- Subject: METABOLISMO
- Language: Português
- Imprenta:
- Source:
- Título do periódico: Programas e Resumos
- Conference title: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular
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ABNT
MINGATTO, Fábio Ermínio et al. Comparison between the effects of nimesulide and its reduced metabolite on mitochondria. 2000, Anais.. São Paulo: SBBq, 2000. . Acesso em: 07 jul. 2022. -
APA
Mingatto, F. E., Santos, A. C. dos, Uyemura, S. A., & Curti, C. (2000). Comparison between the effects of nimesulide and its reduced metabolite on mitochondria. In Programas e Resumos. São Paulo: SBBq. -
NLM
Mingatto FE, Santos AC dos, Uyemura SA, Curti C. Comparison between the effects of nimesulide and its reduced metabolite on mitochondria. Programas e Resumos. 2000 ;[citado 2022 jul. 07 ] -
Vancouver
Mingatto FE, Santos AC dos, Uyemura SA, Curti C. Comparison between the effects of nimesulide and its reduced metabolite on mitochondria. Programas e Resumos. 2000 ;[citado 2022 jul. 07 ] - Mitochondrial dysfunction induced by galangin can lead to cell death
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- Potential mechanisms for mitochondrial Ca+ efflux and permeability transition (MPT) in isolated rat renal cortex mitochondria evaluated with nimesulide, diclofenac sodium and mefenamic acid
- The critical role of mitochondrial energetic impairment in the toxicity of nimesulide of hepatocytes
- Naturally occurring flavonoids protect mitochondria against oxidative stress
- Effects of nimesulide and its reduced metabolite on mitochondria
- The direct NAD(P)H oxidation by the hydroxylated metabolite of nimesulide triggers mitochondrial permeability transition: potential implication on apoptosis induction
- Studies on the relationship between uncoupling-related mitochondrial processes evaluated with nimesulida and mefenamic acid
- Efeito in vitro da fluoxetina e da norfluoxetina em mitocondria de cerebro de rato
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