Potential mechanisms for mitochondrial Ca+ efflux and permeability transition (MPT) in isolated rat renal cortex mitochondria evaluated with nimesulide, diclofenac sodium and mefenamic acid (1999)
- Authors:
- USP affiliated authors: UYEMURA, SERGIO AKIRA - FCFRP ; SANTOS, ANTONIO CARDOZO DOS - FCFRP ; CURTI, CARLOS - FCFRP
- Unidade: FCFRP
- Assunto: BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Source:
- Título: Programa e resumos
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular
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ABNT
PIGOSO, Acácio Antonio et al. Potential mechanisms for mitochondrial Ca+ efflux and permeability transition (MPT) in isolated rat renal cortex mitochondria evaluated with nimesulide, diclofenac sodium and mefenamic acid. 1999, Anais.. Caxambu: SBBq, 1999. . Acesso em: 03 mar. 2026. -
APA
Pigoso, A. A., Tudella, V. G., Uyemura, S. A., Santos, A. C. dos, & Curti, C. (1999). Potential mechanisms for mitochondrial Ca+ efflux and permeability transition (MPT) in isolated rat renal cortex mitochondria evaluated with nimesulide, diclofenac sodium and mefenamic acid. In Programa e resumos. Caxambu: SBBq. -
NLM
Pigoso AA, Tudella VG, Uyemura SA, Santos AC dos, Curti C. Potential mechanisms for mitochondrial Ca+ efflux and permeability transition (MPT) in isolated rat renal cortex mitochondria evaluated with nimesulide, diclofenac sodium and mefenamic acid. Programa e resumos. 1999 ;[citado 2026 mar. 03 ] -
Vancouver
Pigoso AA, Tudella VG, Uyemura SA, Santos AC dos, Curti C. Potential mechanisms for mitochondrial Ca+ efflux and permeability transition (MPT) in isolated rat renal cortex mitochondria evaluated with nimesulide, diclofenac sodium and mefenamic acid. Programa e resumos. 1999 ;[citado 2026 mar. 03 ] - In situ evidence of an alternative oxidase and an uncoupling protein in the respiratory chain of Aspergillus fumigatus
- Diclofenac sodium and mefenamic acid: potent inducers of the membrane permeability transition in renal cortex mitochondria
- Naturally occurring flavonoids protect mitochondria against oxidative stress
- Mitochondrial dysfunction induced by galangin can lead to cell death
- Effects of nimesulide and its reduced metabolite on mitochondria
- Antioxidant activity of flavonoids in isolated mitochondria
- Thioridazine interacts with the membrane of mitochondria acquiring antioxidant activity toward apoptosis - potentially implicated mechanisms
- Structural features improving the antioxidant activity of flavonoids in mitochondria (I): influence of the 2, 3-double bond/4-oxo-function/3-hydroxyl group
- Potential toxicity of toluene and xylene evoked by mitochondrial uncoupling
- The critical role of mitochondrial energetic impairment in the toxicity of nimesulide of hepatocytes
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