Analysis of the involvement of a C-type retrovirus in the glucocorticoid-induced transformed to normal phenotypic reversion displayed by C6/ST1 cells (1998)
- Authors:
- Autor USP: SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; BIOLOGIA MOLECULAR
- Language: Inglês
- Imprenta:
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular
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ABNT
BENGTSON, M. H.; VALENTINI, S R; ARMELIN, Mari Cleide Sogayar. Analysis of the involvement of a C-type retrovirus in the glucocorticoid-induced transformed to normal phenotypic reversion displayed by C6/ST1 cells. Anais.. São Paulo: SBBQ, 1998. -
APA
Bengtson, M. H., Valentini, S. R., & Armelin, M. C. S. (1998). Analysis of the involvement of a C-type retrovirus in the glucocorticoid-induced transformed to normal phenotypic reversion displayed by C6/ST1 cells. In . São Paulo: SBBQ. -
NLM
Bengtson MH, Valentini SR, Armelin MCS. Analysis of the involvement of a C-type retrovirus in the glucocorticoid-induced transformed to normal phenotypic reversion displayed by C6/ST1 cells. 1998 ; -
Vancouver
Bengtson MH, Valentini SR, Armelin MCS. Analysis of the involvement of a C-type retrovirus in the glucocorticoid-induced transformed to normal phenotypic reversion displayed by C6/ST1 cells. 1998 ; - Role of cellular oncogenes in the mechanism of action of platelet - derived growth factor (pdgf)
- Nuclear proto-oncogenes in growth control and in tumor virus-induced neoplasia
- Cultura de celulas
- Isolation and characterization of retinoblastoma tumor cell lines
- Role of growth factor inducible genes in polyoma transformation and growth control
- Effect of polyomavirus middle T antigen on nuclear proto-oncogenes transcription
- Identificacao de mutacoes no gene rb por polimorfismo revelado por pcr e restricao com xbai
- Regulation of the c-fos promoter by pymt antigen
- Glucocorticoid hormones selectively inhibit two growth factor related genes (je and kc) but not the c-myc proto-oncogene
- Molecular basis of glucocos-tiroids action in transformed to normal phenotypic reversion
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