Molecular approach to the anti-tumour activity of glucocorticoides and polyoma transformation (1997)
- Authors:
- Autor USP: SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; GENÉTICA
- Language: Inglês
- Imprenta:
- Publisher: International centre for Genetic Engineering and Biotechnology
- Publisher place: Trieste
- Date published: 1997
- Conference titles: ICGEB Anniversary Symposium
-
ABNT
ARMELIN, Mari Cleide Sogayar et al. Molecular approach to the anti-tumour activity of glucocorticoides and polyoma transformation. 1997, Anais.. Trieste: International centre for Genetic Engineering and Biotechnology, 1997. . Acesso em: 24 abr. 2024. -
APA
Armelin, M. C. S., Vedoy, C., Oliveira, M. L., Carvalho, L. H., Flatschart, R. B., Mercado, J. M., & Alvarenga, R. (1997). Molecular approach to the anti-tumour activity of glucocorticoides and polyoma transformation. In . Trieste: International centre for Genetic Engineering and Biotechnology. -
NLM
Armelin MCS, Vedoy C, Oliveira ML, Carvalho LH, Flatschart RB, Mercado JM, Alvarenga R. Molecular approach to the anti-tumour activity of glucocorticoides and polyoma transformation. 1997 ;[citado 2024 abr. 24 ] -
Vancouver
Armelin MCS, Vedoy C, Oliveira ML, Carvalho LH, Flatschart RB, Mercado JM, Alvarenga R. Molecular approach to the anti-tumour activity of glucocorticoides and polyoma transformation. 1997 ;[citado 2024 abr. 24 ] - Role of polyoma t antigens in balb-c/3t3 cells growth control deregulation
- Further characterization of balb-3t3 transfectants bearing polyoma t antigen genes
- Controle e subversao da proliferacao
- Glucocorticoid-regulated gene in transformed to normal phenotypic reversion
- T antigens role in polyomavirus transformation: c-myc but not c-fos or c-jun expression is a target for middle t
- Cdna cloning and expression of growth control genes
- Fibroblast growth factor and camp induce the expression of fos and jun proto-oncogenes in pc12 cells
- Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma
- Cloning of differentially expressed genes involved glucocorticoid-induced phenotypic reversion of c6 glioma cells
- Hydrazine derivative - induced proliferation superimposed on citotoxity in cultured moused fibroblasts
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