Effects of a new non-steroidal anti-inflammatory drug (nsaid) with the ability to donate nitric oxide (no) on experimental cyclosporine a (csa) acute nephrotoxicity (1996)
- Authors:
- USP affiliated authors: FUJIHARA, CLARICE KAZUE - FM ; ZATZ, ROBERTO - FM ; NUCCI, GILBERTO DE - ICB
- Unidades: FM; ICB
- Assunto: NEFROLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Jornal Brasileiro de Nefrologia
- Volume/Número/Paginação/Ano: v.18, n.1 , p.s22, mar. 1996
- Conference titles: Latin American Congress on Acute Renal Failure
-
ABNT
VIEIRA JUNIOR, J M et al. Effects of a new non-steroidal anti-inflammatory drug (nsaid) with the ability to donate nitric oxide (no) on experimental cyclosporine a (csa) acute nephrotoxicity. Jornal Brasileiro de Nefrologia. São Paulo: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 10 jan. 2026. , 1996 -
APA
Vieira Junior, J. M., Fujihara, C. K., Zatz, R., De Nucci, G., & Burdmann, E. A. (1996). Effects of a new non-steroidal anti-inflammatory drug (nsaid) with the ability to donate nitric oxide (no) on experimental cyclosporine a (csa) acute nephrotoxicity. Jornal Brasileiro de Nefrologia. São Paulo: Faculdade de Medicina, Universidade de São Paulo. -
NLM
Vieira Junior JM, Fujihara CK, Zatz R, De Nucci G, Burdmann EA. Effects of a new non-steroidal anti-inflammatory drug (nsaid) with the ability to donate nitric oxide (no) on experimental cyclosporine a (csa) acute nephrotoxicity. Jornal Brasileiro de Nefrologia. 1996 ;18( 1 ): s22.[citado 2026 jan. 10 ] -
Vancouver
Vieira Junior JM, Fujihara CK, Zatz R, De Nucci G, Burdmann EA. Effects of a new non-steroidal anti-inflammatory drug (nsaid) with the ability to donate nitric oxide (no) on experimental cyclosporine a (csa) acute nephrotoxicity. Jornal Brasileiro de Nefrologia. 1996 ;18( 1 ): s22.[citado 2026 jan. 10 ] - Renal effects of acute and chronic nitric oxide inhibition in experimental diabetes
- Mecanismos de alteracao da permeabilidade glomerular no modelo de inibicao cronica do oxido nitrico (no)
- The inflammatory component in progressive renal disease: are interventions possible
- An extremely high dose of losartan affords superior renoprotection in the remnant model
- Chronic inhibition of nuclear factor-kB attenuates renal injury in the 5/6 renal ablation model
- Effects of an endothelin et receptor antagonist on experimental fk506 induced acute renal dysfunction
- Hypertension and progressive renal injury induced by salt overload in rats preconditioned by temporary nitric oxide (NO) inhibition
- O efeito do tratamento tardio com alta dose de losartan (L) no modelo de ablação renal de 5/6 (NX)
- Extremely high doses of Losartan (L) prevent renal injuryin the remnant kidney model (NX) by an antiinflammatory rather than glomerular hemodynamic effect
- Mechanisms of progressive renal disease: role of angiotensin II, cyclooxygenase products and nitric oxide
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