Expression of growth factors , proto-oncogenes p53 in nasopharyngeal angiofibromas (1996)
- Authors:
- USP affiliated authors: BRENTANI, MARIA MITZI - FM ; BUTUGAN, OSSAMU - FM ; NAGAI, MARIA APARECIDA - FM
- School: FM
- Subject: ONCOLOGIA
- Language: Inglês
- Imprenta:
- Place of publication: Saint Louis
- Date published: 1996
- Source:
- Título do periódico: Laryngoscope
- Volume/Número/Paginação/Ano: v.106, n.2 pt.1, p.190-5, feb. 1996
-
ABNT
NAGAI, Maria Aparecida; BUTUGAN, Ossamu; BRENTANI, Maria Mitzi. Expression of growth factors , proto-oncogenes p53 in nasopharyngeal angiofibromas. Laryngoscope, Saint Louis, v. 106, n. 2 pt.1, p. 190-5, 1996. -
APA
Nagai, M. A., Butugan, O., & Brentani, M. M. (1996). Expression of growth factors , proto-oncogenes p53 in nasopharyngeal angiofibromas. Laryngoscope, 106( 2 pt.1), 190-5. -
NLM
Nagai MA, Butugan O, Brentani MM. Expression of growth factors , proto-oncogenes p53 in nasopharyngeal angiofibromas. Laryngoscope. 1996 ;106( 2 pt.1): 190-5. -
Vancouver
Nagai MA, Butugan O, Brentani MM. Expression of growth factors , proto-oncogenes p53 in nasopharyngeal angiofibromas. Laryngoscope. 1996 ;106( 2 pt.1): 190-5. - Relaxation of imprinting of IGFII gene in juvenile nasopharyngeal angiofibromas
- C-KI-RAS and C-HA-RAS mutations are rare whereas C-HA-RAS overexpression is frequently observed in head and neck cancer
- Alterações no gene P16 em nasoangiofibroma juvenil
- Relaxation of imprinting of IGFII gene in juvenile nasopharyngeal angiofibromas
- Loss of imprinting of IGFI gene in juvenile nasopharyngeal angiofibromas
- Genetic alterations in Ki-ras and Ha-ras genes in juvenile nasopharyngeal angiofibromas and head and neck cancer
- A expressão do protooncogene C-MYC na próstata humana é modulada pela ação de androgênios
- Overexpression of the c-myc proto-oncogene in breast cancer
- Gene expression profile of estrogen receptor negative breast cancer cells transfected with cDNA for estrogen receptor
- Tumor-associated genes differentially expressed in breast tumors regarding the presence or absence of estrogen and progesterone receptors
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