Trypanosoma cruzi: il-10, tnf, ifn-gama, and il-12 regulate innate and acquired immunity to infection (1996)
- Authors:
- Autor USP: ABRAHAMSOHN, ISES DE ALMEIDA - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Source:
- Título do periódico: Experimental Parasitology
- Volume/Número/Paginação/Ano: v.84, p.231-44, 1996
-
ABNT
ABRAHAMSOHN, Ises de Almeida e COFFMAN, R L. Trypanosoma cruzi: il-10, tnf, ifn-gama, and il-12 regulate innate and acquired immunity to infection. Experimental Parasitology, v. 84, p. 231-44, 1996Tradução . . Acesso em: 23 abr. 2024. -
APA
Abrahamsohn, I. de A., & Coffman, R. L. (1996). Trypanosoma cruzi: il-10, tnf, ifn-gama, and il-12 regulate innate and acquired immunity to infection. Experimental Parasitology, 84, 231-44. -
NLM
Abrahamsohn I de A, Coffman RL. Trypanosoma cruzi: il-10, tnf, ifn-gama, and il-12 regulate innate and acquired immunity to infection. Experimental Parasitology. 1996 ;84 231-44.[citado 2024 abr. 23 ] -
Vancouver
Abrahamsohn I de A, Coffman RL. Trypanosoma cruzi: il-10, tnf, ifn-gama, and il-12 regulate innate and acquired immunity to infection. Experimental Parasitology. 1996 ;84 231-44.[citado 2024 abr. 23 ] - Endogenously produced IL-10 and prostaglandins regulate peripheral blood mononuclear cells proliferation to Trypanosoma cruzi antigens
- Nitric oxide produced by macrophages and regulation of resistance to "Trypanosoma cruzi" at the initial phase of infection
- Egulation of Trypanosoma cruzi infection by type interferons
- Il-12 regulation of ifn-gama synthesis in the acute phase of trypanosoma cruzi infection
- IL-12 regulation of IFN-'gama' synthesis in the acute phase of Trypanosoma cruzi infection
- Prostaglandins mediate suppression of lymphoproliferation and cytokine synthesis during the early phase of Trypanosoma cruzi infection
- Il-12 enhances proliferation of peripheral blood mononuclear cells from chagasic patients to trypanosoma cruzi antigen
- Il-10 and ifn-gamma production by spleen cells in trypanosoma cruzi infected mice
- Bordetella pertussis and innate immune response: inhibition of NO production by murine BMDMO through TGF-ß arginase and TLR4
- Immature macrophages derived from mouse bone marrow produce large amounts of IL-12p40 after LPS stimulation
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