Sodium excess aggravates hypertension and renal parenchymal injury in rats with chronic no inhibition (1994)
- Authors:
- USP affiliated authors: FUJIHARA, CLARICE KAZUE - FM ; ZATZ, ROBERTO - FM
- School: FM
- DOI: 10.1152/ajprenal.1994.266.5.f697
- Subject: NEFROLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: American Journal of Physiology
- Volume/Número/Paginação/Ano: v.35, n.5 , p.f697-705, mai. 1994
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
FUJIHARA, C K; MICHELLAZZO, S M; DE NUCCI, Gilberto; ZAIZ, R. Sodium excess aggravates hypertension and renal parenchymal injury in rats with chronic no inhibition. American Journal of Physiology, Bethesda, v. 35, n. 5 , p. f697-705, 1994. DOI: 10.1152/ajprenal.1994.266.5.f697. -
APA
Fujihara, C. K., Michellazzo, S. M., De Nucci, G., & Zaiz, R. (1994). Sodium excess aggravates hypertension and renal parenchymal injury in rats with chronic no inhibition. American Journal of Physiology, 35( 5 ), f697-705. doi:10.1152/ajprenal.1994.266.5.f697 -
NLM
Fujihara CK, Michellazzo SM, De Nucci G, Zaiz R. Sodium excess aggravates hypertension and renal parenchymal injury in rats with chronic no inhibition. American Journal of Physiology. 1994 ;35( 5 ): f697-705. -
Vancouver
Fujihara CK, Michellazzo SM, De Nucci G, Zaiz R. Sodium excess aggravates hypertension and renal parenchymal injury in rats with chronic no inhibition. American Journal of Physiology. 1994 ;35( 5 ): f697-705. - Bloqueio cronico do oxido nitrico agrava a glomerulosclerose associada a ablacao renal
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- Physiologic role of endothelin on regulation of renal and systemic hemodynamics in the rat
- Bloqueio do oxido nitrico reverte a hiperfiltracao glomerular em ratos diabeticos
- Reversão da atividade inflamatória renal no modelo de inibição crônica do óxido nítrico
- Losartan-hydrochlorothiazide association promotes lasting blood pressure normalization and completely arrests long-term renal injury in the 5/6 ablation model
- Role of glomerular hypertension: glomerular hypertrophy and lipid deposition in the genesis of glomerular sclerosis (gs) of experimental diabetes (dm)
- Absence of focal glomerulosclerosis in aging analbuminemic rats
- Effects of an endothelin et receptor antagonist on experimental fk506 induced acute renal dysfunction
- Massive albuminuria glomerulosclerosis (gs) by chronic nitric oxide (no) blackade and sodium overload
Informações sobre o DOI: 10.1152/ajprenal.1994.266.5.f697 (Fonte: oaDOI API)
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