Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma (1994)
- Authors:
- Autor USP: SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- Assunto: BIOLOGIA MOLECULAR
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquimica e Biologia Molecular
- Publisher place: São Paulo
- Date published: 1994
- Source:
- Título do periódico: Programa e Resumos
- Conference titles: Reuniao Anual da Sociedade Brasileira de Bioquimica e Biologia Molecular
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ABNT
VALENTINI, S R; OLIVEIRA, M. L. S.; SASAHARA, R M; ARMELIN, M C S. Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma. Anais.. São Paulo: Sociedade Brasileira de Bioquimica e Biologia Molecular, 1994. -
APA
Valentini, S. R., Oliveira, M. L. S., Sasahara, R. M., & Armelin, M. C. S. (1994). Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma. In Programa e Resumos. São Paulo: Sociedade Brasileira de Bioquimica e Biologia Molecular. -
NLM
Valentini SR, Oliveira MLS, Sasahara RM, Armelin MCS. Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma. Programa e Resumos. 1994 ; -
Vancouver
Valentini SR, Oliveira MLS, Sasahara RM, Armelin MCS. Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma. Programa e Resumos. 1994 ; - Role of cellular oncogenes in the mechanism of action of platelet - derived growth factor (pdgf)
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- Cultura de celulas
- Isolation and characterization of retinoblastoma tumor cell lines
- Role of growth factor inducible genes in polyoma transformation and growth control
- Effect of polyomavirus middle T antigen on nuclear proto-oncogenes transcription
- Identificacao de mutacoes no gene rb por polimorfismo revelado por pcr e restricao com xbai
- Regulation of the c-fos promoter by pymt antigen
- Glucocorticoid hormones selectively inhibit two growth factor related genes (je and kc) but not the c-myc proto-oncogene
- Molecular basis of glucocos-tiroids action in transformed to normal phenotypic reversion
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