Regulation of the glucocorticoid receptor gene promoter by ap-1 family members (1994)
- Authors:
- USP affiliated authors: BRENTANI, MARIA MITZI - FM ; MARTINS, VILMA REGINA - FM
- Unidade: FM
- Assunto: ONCOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquimica e Biologia Molecular
- Publisher place: Sao Paulo
- Date published: 1994
- Source:
- Título do periódico: Programa e Resumos
- Conference titles: Reuniao Anual da Sociedade Brasileira de Bioquimica e Biologia Molecular
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ABNT
MARTINS, Vilma Regina et al. Regulation of the glucocorticoid receptor gene promoter by ap-1 family members. 1994, Anais.. Sao Paulo: Sociedade Brasileira de Bioquimica e Biologia Molecular, 1994. . Acesso em: 29 mar. 2024. -
APA
Martins, V. R., Cabral, A. L. B., Araujo, A. B., Housley, P. R., & Brentani, M. M. (1994). Regulation of the glucocorticoid receptor gene promoter by ap-1 family members. In Programa e Resumos. Sao Paulo: Sociedade Brasileira de Bioquimica e Biologia Molecular. -
NLM
Martins VR, Cabral ALB, Araujo AB, Housley PR, Brentani MM. Regulation of the glucocorticoid receptor gene promoter by ap-1 family members. Programa e Resumos. 1994 ;[citado 2024 mar. 29 ] -
Vancouver
Martins VR, Cabral ALB, Araujo AB, Housley PR, Brentani MM. Regulation of the glucocorticoid receptor gene promoter by ap-1 family members. Programa e Resumos. 1994 ;[citado 2024 mar. 29 ] - Attenuation of glucocorticoid receptor levels by the h-ras oncogene
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- Influence of oncogenes on glucocorticoid receptor binding activity
- Alteration of c-jun and c-fos mrna levels in cell lines with low glucocorticoid receptor gene expression
- Glucocorticoid receptors of lymphocites from myastenia gravis patients
- Steroid receptors in the myasthenic thymus
- Fibrosarcoma - ras: a morphometrical and immunohistochemical study
- Cellular transformation with ha-ras oncogene involves decrease in glucocorticoid receptor gene expression
- Effect of ej-ras transformation on the glucocorticoid receptor
- Identification of the glucorticoid receptor gene promoter regions mediating the basal activity and the regulation by glucorticoids
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