Isolation and characterization of v-79 chinese hamster cell clones transfected with transferrin receptor c-dna lacking the iron regulatory domain (1993)
- Authors:
- Autor USP: MENEGHINI, ROGERIO - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; BIOLOGIA MOLECULAR
- Language: Inglês
- Imprenta:
- Publisher: Brazilian Society of Orthomolecular Medicine
- Publisher place: São Paulo
- Date published: 1993
- Source:
- Título do periódico: Program and Abstracts
- Conference titles: International Symposium on Orthomolecular Medicine
-
ABNT
NASCIMENTO, A L T O e SEBASTIAO, P C A e MENEGHINI, R. Isolation and characterization of v-79 chinese hamster cell clones transfected with transferrin receptor c-dna lacking the iron regulatory domain. 1993, Anais.. São Paulo: Brazilian Society of Orthomolecular Medicine, 1993. . Acesso em: 25 abr. 2024. -
APA
Nascimento, A. L. T. O., Sebastiao, P. C. A., & Meneghini, R. (1993). Isolation and characterization of v-79 chinese hamster cell clones transfected with transferrin receptor c-dna lacking the iron regulatory domain. In Program and Abstracts. São Paulo: Brazilian Society of Orthomolecular Medicine. -
NLM
Nascimento ALTO, Sebastiao PCA, Meneghini R. Isolation and characterization of v-79 chinese hamster cell clones transfected with transferrin receptor c-dna lacking the iron regulatory domain. Program and Abstracts. 1993 ;[citado 2024 abr. 25 ] -
Vancouver
Nascimento ALTO, Sebastiao PCA, Meneghini R. Isolation and characterization of v-79 chinese hamster cell clones transfected with transferrin receptor c-dna lacking the iron regulatory domain. Program and Abstracts. 1993 ;[citado 2024 abr. 25 ] - Ciencia e universidade publica
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- Evidence for the mechanism of direct attack by hydroxyl radical on dna from mammalian cells under oxidative stress
- Possible involvement of intracellular iron in dna damage by oxidative stress in v-79 chinese hamster transfected cells with human transferrin receptor c-dna
- Iron metabolism and dna damage
- Activation of iron responsive element-binding protein by oxidative stress in mammalian cells
- Respiracao produz compostos prejudiciais a saude
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