Is inf-alpha involved in early susceptibility of Trypanosoma cruzi infected C3H / he mice? (1991)
- Authors:
- USP affiliated authors: STAROBINAS, NANCY - ICB ; RUSSO, MOMTCHILO - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Research in Immunology
- Volume/Número/Paginação/Ano: v. 142, n. 2, p. 117-122, 1991
-
ABNT
STAROBINAS, Nancy et al. Is inf-alpha involved in early susceptibility of Trypanosoma cruzi infected C3H / he mice?. Research in Immunology, v. 142, n. 2, p. 117-122, 1991Tradução . . Acesso em: 14 fev. 2026. -
APA
Starobinas, N., Russo, M., Minoprio, P., & Hontebeyrie-Joskowicz, M. (1991). Is inf-alpha involved in early susceptibility of Trypanosoma cruzi infected C3H / he mice? Research in Immunology, 142( 2), 117-122. -
NLM
Starobinas N, Russo M, Minoprio P, Hontebeyrie-Joskowicz M. Is inf-alpha involved in early susceptibility of Trypanosoma cruzi infected C3H / he mice? Research in Immunology. 1991 ; 142( 2): 117-122.[citado 2026 fev. 14 ] -
Vancouver
Starobinas N, Russo M, Minoprio P, Hontebeyrie-Joskowicz M. Is inf-alpha involved in early susceptibility of Trypanosoma cruzi infected C3H / he mice? Research in Immunology. 1991 ; 142( 2): 117-122.[citado 2026 fev. 14 ] - Susceptible mice present higher macrophage activation than resistant mice during infections with myotropic strains of Trypanosoma cruzi
- Comparison of microphage activation induced in vivo and in vitro by glucose / mannose binding plant lectins
- Peritoneal macrophages stimulated in vivo and in vitro by plant lectins differin their expression of class II mhc molecules and nitric oxide production
- MHC class II expression nitric oxide (No) release and ifn-gama induced no in cells of susceptible T. Cruzi infected mice
- Anticorpo monoclonal anti mhc de classe II impede a indução de tolerância por via oral a ovoalbumina
- Interactions between PAF and TNF in immune-complex arthritis in rats
- Activation and priming of macrophages for nitric oxide release in susceptible and resistant mice during trypanosoma cruzi infection
- Parasitic load increases and myocardial inflammation decreases in Trypanosoma cruzi: infected mice after inactivation of helper T cells
- Control of Trypanosoma cruzi multiplication by t cell subsets in different tissues
- Macrophage activation and resistance to Trypanosoma cruzi infection
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