Ha-ras-1 oncogene dosage differentially affects balb / 3t3 cells , growth factor requirement and tumorigenicity (1989)
- Authors:
- USP affiliated authors: ARMELIN, HUGO AGUIRRE - IQ ; SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- Assunto: ONCOGENES
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Oncogene Research
- Volume/Número/Paginação/Ano: v. 4, n. 1, p. 55-64, 1989
-
ABNT
KOVARY, K e ARMELIN, M. C. S e ARMELIN, Hugo Aguirre. Ha-ras-1 oncogene dosage differentially affects balb / 3t3 cells , growth factor requirement and tumorigenicity. Oncogene Research, v. 4, n. 1, p. 55-64, 1989Tradução . . Acesso em: 19 abr. 2024. -
APA
Kovary, K., Armelin, M. C. S., & Armelin, H. A. (1989). Ha-ras-1 oncogene dosage differentially affects balb / 3t3 cells , growth factor requirement and tumorigenicity. Oncogene Research, 4( 1), 55-64. -
NLM
Kovary K, Armelin MCS, Armelin HA. Ha-ras-1 oncogene dosage differentially affects balb / 3t3 cells , growth factor requirement and tumorigenicity. Oncogene Research. 1989 ; 4( 1): 55-64.[citado 2024 abr. 19 ] -
Vancouver
Kovary K, Armelin MCS, Armelin HA. Ha-ras-1 oncogene dosage differentially affects balb / 3t3 cells , growth factor requirement and tumorigenicity. Oncogene Research. 1989 ; 4( 1): 55-64.[citado 2024 abr. 19 ] - Generation of myc / fos transfectant bolb - 3t3 cell line
- Mutated ej-c-ha-ras-l human oncogene causes malignant transformation without abolishing growth requirement for fgf and egf
- Fudr selection of revertants from ej-ras-transformed 3t3 cells
- Mode of v-fos oncogene integration in 3t3 cells genome
- Isolation and characterization of balb/c double transfectant cell lines contitutively expressing c-myc and v-fos oncogenes
- Mechanisms of action of growth factors and oncogene expression
- Glucocorticoid dexamethasone reversibly complements 'E IND.J'-ras oncogene to transform mouse embryo balb - 3t3 cells
- Role played by oncogene products in peptide growth factor's action
- Interactions of peptide growth factors and oncogenes
- Interaction between myc and fos oncogenes in the control of cell proliferation
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