Long-term survival of mouse pancreatic islets in vitro following acute exposure to streptozotocin altered insulin response to scretagojures (1987)
- Authors:
- Autor USP: EIZIRIK, DECIO LAKS - FMRP
- Unidade: FMRP
- Subjects: DIABETES MELLITUS; INSULINA; EXPERIMENTOS ANIMAIS
- Language: Português
- Source:
- Título: Diabetologia
- Volume/Número/Paginação/Ano: v.30, p.516, 1987
-
ABNT
EIZIRIK, D L e STRABDELL, E e SANDLER, S. Long-term survival of mouse pancreatic islets in vitro following acute exposure to streptozotocin altered insulin response to scretagojures. Diabetologia, v. 30, p. 516, 1987Tradução . . Acesso em: 25 jan. 2026. -
APA
Eizirik, D. L., Strabdell, E., & Sandler, S. (1987). Long-term survival of mouse pancreatic islets in vitro following acute exposure to streptozotocin altered insulin response to scretagojures. Diabetologia, 30, 516. -
NLM
Eizirik DL, Strabdell E, Sandler S. Long-term survival of mouse pancreatic islets in vitro following acute exposure to streptozotocin altered insulin response to scretagojures. Diabetologia. 1987 ;30 516.[citado 2026 jan. 25 ] -
Vancouver
Eizirik DL, Strabdell E, Sandler S. Long-term survival of mouse pancreatic islets in vitro following acute exposure to streptozotocin altered insulin response to scretagojures. Diabetologia. 1987 ;30 516.[citado 2026 jan. 25 ] - 1.10 phenantroline , a metal chelator, protectes against alloxan but not streptozotocin induced eytotoxicity , in human fibroblosts and rat cells
- Functional characteristics of cultured mouse pancreatic islets following exposure to different streptozotocin concentrations
- Effects of metal chilator (1-10 phenantroline) on streptozotocin and alloxan-induced diabets in the rat
- Studies on the mechanism causing the impairment of glucose - stimulated insulin release in rat pancreatic islets exposed to human recombinant interleukin 1 beta
- Efeito de uma dieta hiperproteica sobre a acao diabetogenica da estreptozotocina em ratos
- Partial protective effect of branched chain amino acids against streptozotocin induced cytotoxicity to mouse pancreatic islets on vitro
- Heat shock treatment of mouse pancreatic islets cells but no remaining functional impairment among the surviving beta-cells
- Preferential reduction of insulim production in mouse pancreatic islets maintained in culture after streptozotocin exposure
- Defective carbolism of d. Glucose and l. Glutamine in mouse pancreatic islets maintained in culture after streptozotocin exposure
- Studies on the mechanisms causing inhibition of inclulin secretion in rat pancreatic islets exposed to human interlenkin 1 b indicate a perturbation in the mitochondrial function
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