Role of the fungal cell wall in the granulomatous response of mice to the agents of chromoblasto mycosis (1985)
- Authors:
- Autor USP: SILVA, CELIO LOPES - FMRP
- Unidade: FMRP
- Subjects: CROMOBLASTOMICOSE; VIRULÊNCIA
- Language: Português
- Source:
- Título do periódico: Journal of Medical Microbiology
- Volume/Número/Paginação/Ano: v.20, p.299-305, 1985
-
ABNT
SILVA, C L; FAZIOLI, R A. Role of the fungal cell wall in the granulomatous response of mice to the agents of chromoblasto mycosis. Journal of Medical Microbiology[S.l.], v. 20, p. 299-305, 1985. -
APA
Silva, C. L., & Fazioli, R. A. (1985). Role of the fungal cell wall in the granulomatous response of mice to the agents of chromoblasto mycosis. Journal of Medical Microbiology, 20, 299-305. -
NLM
Silva CL, Fazioli RA. Role of the fungal cell wall in the granulomatous response of mice to the agents of chromoblasto mycosis. Journal of Medical Microbiology. 1985 ;20 299-305. -
Vancouver
Silva CL, Fazioli RA. Role of the fungal cell wall in the granulomatous response of mice to the agents of chromoblasto mycosis. Journal of Medical Microbiology. 1985 ;20 299-305. - Cell wall components of ac actinomycetis involved in the pathogeny of actinomicotic mycetona
- Cord factor is associated with the maintenance of chronic inflammatory reaction caused by mycobacteria
- Mouse toxicity induced by lipids and cell wall isolated from actinomycetis
- Participation of tumor necrosis factor in the anti-tumor activity of mycobacterial trehalose dimycolate (cord factor)
- Endogenous presentation of m. Leprae hsp65 antigen by transfected macrophages to cd4+ - specific t lymphocytes
- Vacinacao de camundongos com macrofagos transfectados e expressando hsp65
- Transfeccao de macrofagos com genes de mycobacterium lepraes
- Transfeccao de macrofagos com genes de proteinas imunogenicas de m. Leprae
- Mycobacterium leprae g5hsp antigen expressed from a retroviral vector in a macrophage cell line is presented to t cells in association with class ii in addition to mmc class i
- Tnf and macrophage activation are essential for host resistance to nocardia brasilienses infection
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