ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma (2018)
- Autores:
- Autores USP: CRUZ, MARIO COSTA - ICB ; RIBEIRO, PRISCILLA RAMOS LARA - ICB ; JAEGER, RUY GASTALDONI - ICB ; FREITAS, VANESSA MORAIS - ICB
- Unidade: ICB
- DOI: 10.1016/j.yexcr.2018.01.017
- Assuntos: FIBROSSARCOMA; PROLIFERAÇÃO CELULAR; MIGRAÇÃO; BIOLOGIA CELULAR
- Agências de fomento:
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Experimental Cell Research
- ISSN: 1090-2422
- Volume/Número/Paginação/Ano: v. 363, n. 2, p. 271-282, 2018
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
GUERRA, Heydi Noriega et al. ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma. Experimental Cell Research, v. 363, n. 2, p. 271-282, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.yexcr.2018.01.017. Acesso em: 16 abr. 2024. -
APA
Guerra, H. N., Cruz, M. C., Ribeiro, P. R. L., Strnadel, J., Wang, H., Klemke, R. L., et al. (2018). ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma. Experimental Cell Research, 363( 2), 271-282. doi:10.1016/j.yexcr.2018.01.017 -
NLM
Guerra HN, Cruz MC, Ribeiro PRL, Strnadel J, Wang H, Klemke RL, Jaeger RG, Freitas VM. ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma [Internet]. Experimental Cell Research. 2018 ; 363( 2): 271-282.[citado 2024 abr. 16 ] Available from: https://doi.org/10.1016/j.yexcr.2018.01.017 -
Vancouver
Guerra HN, Cruz MC, Ribeiro PRL, Strnadel J, Wang H, Klemke RL, Jaeger RG, Freitas VM. ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma [Internet]. Experimental Cell Research. 2018 ; 363( 2): 271-282.[citado 2024 abr. 16 ] Available from: https://doi.org/10.1016/j.yexcr.2018.01.017 - AHNAK enables mammary carcinoma cells to produce extracellular vesicles that increase neighboring fibroblast cell motility
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Informações sobre o DOI: 10.1016/j.yexcr.2018.01.017 (Fonte: oaDOI API)
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