Exploring IKKβ as an anti-metastatic therapeutic target in KRAS-induced lung cancer (2017)
- Authors:
- Autor USP: BASSÈRES, DANIELA SANCHEZ - IQ
- Unidade: IQ
- Subjects: NEOPLASIAS PULMONARES; MUTAÇÃO
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq
- Publisher place: São Paulo
- Date published: 2017
- Source:
- Título do periódico: Abstracts
- Conference titles: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology
-
ABNT
MIRANDA, Vanessa Silva e BASSÈRES, Daniela Sanchez e CARNEIRO LOBO, Tatiana Correa. Exploring IKKβ as an anti-metastatic therapeutic target in KRAS-induced lung cancer. 2017, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq, 2017. . Acesso em: 24 abr. 2024. -
APA
Miranda, V. S., Bassères, D. S., & Carneiro Lobo, T. C. (2017). Exploring IKKβ as an anti-metastatic therapeutic target in KRAS-induced lung cancer. In Abstracts. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq. -
NLM
Miranda VS, Bassères DS, Carneiro Lobo TC. Exploring IKKβ as an anti-metastatic therapeutic target in KRAS-induced lung cancer. Abstracts. 2017 ;[citado 2024 abr. 24 ] -
Vancouver
Miranda VS, Bassères DS, Carneiro Lobo TC. Exploring IKKβ as an anti-metastatic therapeutic target in KRAS-induced lung cancer. Abstracts. 2017 ;[citado 2024 abr. 24 ] - Aurora kinases: potential therapeutic targets in K-Ras-induced lung cancer
- Targeted BMI1 inhibition impairs tumor growth in lung adenocarcinomas with low CEBP alpha expression
- Exploring IKKβ kinase as a therapeutic target for KRAS-driven lung tumor initiating cells
- Targeting Aurora A kinase activity in lung cancer initiating cells driven by oncogenic KRAS
- IKKβ inhibition suppresses sphere formation and self renewal of lung cancer initiating cells
- Aurora kinase targeting in lung cancer reduces KRAS-induced transformation
- IKKβ kinase promotes stemness, migration, and invasion in KRAS-driven lung adenocarcinoma cells
- Identification of a targetable KRAS-mutant epithelial population in non-small cell lung cancer
- FOXO1 inhibition by oncogenic KRAS as an important mechanism promoting lung oncogenesis
- Requirement of the NF-'capa' B subunit p65/RelA for 'capa'-Ras-induced lung tumorigenesis
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