Bicarbonate increases Ischemia-Reperfusion damage by inhibiting mitophagy (2016)
- Authors:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- DOI: 10.1371/journal.pone.0167678
- Subjects: ISQUEMIA; MITOCÔNDRIAS
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2016
- Source:
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
QUELICONI, Bruno Barros e KOWALTOWSKI, Alicia Juliana e GOTTLIEB, Roberta A. Bicarbonate increases Ischemia-Reperfusion damage by inhibiting mitophagy. PLOS ONE, v. 11, n. 12, p. 1-17 art. e0167678, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0167678. Acesso em: 28 mar. 2024. -
APA
Queliconi, B. B., Kowaltowski, A. J., & Gottlieb, R. A. (2016). Bicarbonate increases Ischemia-Reperfusion damage by inhibiting mitophagy. PLOS ONE, 11( 12), 1-17 art. e0167678. doi:10.1371/journal.pone.0167678 -
NLM
Queliconi BB, Kowaltowski AJ, Gottlieb RA. Bicarbonate increases Ischemia-Reperfusion damage by inhibiting mitophagy [Internet]. PLOS ONE. 2016 ; 11( 12): 1-17 art. e0167678.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1371/journal.pone.0167678 -
Vancouver
Queliconi BB, Kowaltowski AJ, Gottlieb RA. Bicarbonate increases Ischemia-Reperfusion damage by inhibiting mitophagy [Internet]. PLOS ONE. 2016 ; 11( 12): 1-17 art. e0167678.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1371/journal.pone.0167678 - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
Informações sobre o DOI: 10.1371/journal.pone.0167678 (Fonte: oaDOI API)
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