Enhanced nitric oxide generation from nitric oxide synthases as the cause of increased peroxynitrite formation during acute restraint stress: Effects on carotid responsiveness to angiotensinergic stimuli in type-1 diabetic rats (2016)
- Autores:
- Autores USP: SILVA, CARLOS HENRIQUE TOMICH DE PAULA DA - FCFRP ; GOMES, MAYARA SANTOS - FCFRP ; OLIVEIRA, ANA MARIA DE - FCFRP
- Unidade: FCFRP
- DOI: 10.1016/j.ejphar.2016.04.050
- Assuntos: SISTEMA RENINA-ANGIOTENSINA; ÓXIDO NÍTRICO; DIABETES MELLITUS; ESTRESSE OXIDATIVO
- Palavras-chave do autor: Type-1 Diabetes; Acute restraint stress; Reactive oxygen species; Reactive nitrogen species; Nitric oxide synthases; Renin-angiotensin-system
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: European Journal of Pharmacology
- ISSN: 0014-2999
- Volume/Número/Paginação/Ano: v. 783, p. 11-22, 2016
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: unspecified-oa
-
ABNT
MOREIRA, Josimar D et al. Enhanced nitric oxide generation from nitric oxide synthases as the cause of increased peroxynitrite formation during acute restraint stress: Effects on carotid responsiveness to angiotensinergic stimuli in type-1 diabetic rats. European Journal of Pharmacology, v. 783, p. 11-22, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2016.04.050. Acesso em: 19 set. 2024. -
APA
Moreira, J. D., Pernomian, L., Gomes, M. S., Moreira, R. P., Prado, A. F. do, Silva, C. H. T. de P. da, & Oliveira, A. M. de. (2016). Enhanced nitric oxide generation from nitric oxide synthases as the cause of increased peroxynitrite formation during acute restraint stress: Effects on carotid responsiveness to angiotensinergic stimuli in type-1 diabetic rats. European Journal of Pharmacology, 783, 11-22. doi:10.1016/j.ejphar.2016.04.050 -
NLM
Moreira JD, Pernomian L, Gomes MS, Moreira RP, Prado AF do, Silva CHT de P da, Oliveira AM de. Enhanced nitric oxide generation from nitric oxide synthases as the cause of increased peroxynitrite formation during acute restraint stress: Effects on carotid responsiveness to angiotensinergic stimuli in type-1 diabetic rats [Internet]. European Journal of Pharmacology. 2016 ; 783 11-22.[citado 2024 set. 19 ] Available from: https://doi.org/10.1016/j.ejphar.2016.04.050 -
Vancouver
Moreira JD, Pernomian L, Gomes MS, Moreira RP, Prado AF do, Silva CHT de P da, Oliveira AM de. Enhanced nitric oxide generation from nitric oxide synthases as the cause of increased peroxynitrite formation during acute restraint stress: Effects on carotid responsiveness to angiotensinergic stimuli in type-1 diabetic rats [Internet]. European Journal of Pharmacology. 2016 ; 783 11-22.[citado 2024 set. 19 ] Available from: https://doi.org/10.1016/j.ejphar.2016.04.050 - Acute restraint stress increases carotid reactivity in type-I diabetic rats by enhancing Nox4/NADPH oxidase functionality
- Pharmacological significance of the interplay between angiotensin receptors: MAS receptors as putative final mediators of the effects elicited by angiotensin AT1 receptors antagonists
- Endothelial oxidative stress induced by diabetes mellitus I increases maximum contraction evoked by angiotensin II in rat carotid artery
- Consequências do tratamento moderadamente colesterolêmico iniciado em camundongos C57BL/6 jovens na contração de ANG II em aorta torácica
- MAS receptors mediate vasoprotective and atheroprotective effects of candesartan upon the recovery of vascular angiotensin-converting enzyme 2–angiotensin-(1-7)–MAS axis functionality
- Mild cholesterolemic diet-elicited oxidative stress impairs the endhothelial relaxation evoked by angiotensin-(1-7) in adult C57CL/6 mice thoracic aorta
- Cross-talk with 'beta IND.2'-adrenoceptors enchances ligand affinity properties from endothelial 'alpha IND.1D'-adrenoceptors that mediates carotid relaxation
- Mas-mediated antioxidant effects restore the functionality of angiotensin converting enzyme 2-angiotensin-(1–7)-mas axis in diabetic rat carotid
- Consequências da lesão por catéter-balão sobre o relaxamento induzido por ANG-(1-7) em carótida contra-lateral de rato diabético tipo I
- Diabetes tipo-I e os eixos ECA- ANG II -AT1 e ECA2-ANG-(1-7)-mas do sistema angiotensinérgico de rato: consequências do estresse de restrição
Informações sobre o DOI: 10.1016/j.ejphar.2016.04.050 (Fonte: oaDOI API)
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