Vinpocetine reduces lipopolysaccharide-induced inflammatory pain and neutrophil recruitment in mice by targeting oxidative stress, cytokines and NF-κB (2015)
- Autores:
- Ruiz-Miyazawa, Kenji W. - Universidade Estadual de Londrina (UEL)
- Pinho-Ribeiro, Felipe A. - Universidade Estadual de Londrina (UEL)
- Zarpelon, Ana C. - Universidade Estadual de Londrina (UEL)
- Staurengo-Ferrari, Larissa - Universidade Estadual de Londrina (UEL)
- Silva, Rangel L.
- Alves Filho, José Carlos Farias
- Cunha, Thiago Mattar
- Cunha, Fernando de Queiroz
- Casagrande, Rúbia - Universidade Estadual de Londrina (UEL)
- Verri Júnior, Waldiceu Aparecido - Universidade Estadual de Londrina (UEL)
- Autores USP: ALVES FILHO, JOSÉ CARLOS FARIAS - FMRP ; CUNHA, THIAGO MATTAR - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidade: FMRP
- DOI: 10.1016/j.cbi.2015.05.007
- Assuntos: CITOCINAS; LIPOPOLISSACARÍDEOS; MACRÓFAGOS; NEUTRÓFILOS; CAVIDADE PERITONEAL; ESTRESSE OXIDATIVO
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Chemico-Biological Interactions
- ISSN: 0009-2797
- Volume/Número/Paginação/Ano: v. 237, p. 9-17, 2015
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: unspecified-oa
-
ABNT
RUIZ-MIYAZAWA, Kenji W. et al. Vinpocetine reduces lipopolysaccharide-induced inflammatory pain and neutrophil recruitment in mice by targeting oxidative stress, cytokines and NF-κB. Chemico-Biological Interactions, v. 237, p. 9-17, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2015.05.007. Acesso em: 25 set. 2024. -
APA
Ruiz-Miyazawa, K. W., Pinho-Ribeiro, F. A., Zarpelon, A. C., Staurengo-Ferrari, L., Silva, R. L., Alves Filho, J. C. F., et al. (2015). Vinpocetine reduces lipopolysaccharide-induced inflammatory pain and neutrophil recruitment in mice by targeting oxidative stress, cytokines and NF-κB. Chemico-Biological Interactions, 237, 9-17. doi:10.1016/j.cbi.2015.05.007 -
NLM
Ruiz-Miyazawa KW, Pinho-Ribeiro FA, Zarpelon AC, Staurengo-Ferrari L, Silva RL, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Vinpocetine reduces lipopolysaccharide-induced inflammatory pain and neutrophil recruitment in mice by targeting oxidative stress, cytokines and NF-κB [Internet]. Chemico-Biological Interactions. 2015 ; 237 9-17.[citado 2024 set. 25 ] Available from: https://doi.org/10.1016/j.cbi.2015.05.007 -
Vancouver
Ruiz-Miyazawa KW, Pinho-Ribeiro FA, Zarpelon AC, Staurengo-Ferrari L, Silva RL, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Vinpocetine reduces lipopolysaccharide-induced inflammatory pain and neutrophil recruitment in mice by targeting oxidative stress, cytokines and NF-κB [Internet]. Chemico-Biological Interactions. 2015 ; 237 9-17.[citado 2024 set. 25 ] Available from: https://doi.org/10.1016/j.cbi.2015.05.007 - Differential regulation of oxidative stress and cytokine production by endothelin ETA and ETB receptors in superoxide anion-induced inflammation and pain in mice
- Bosentan, a mixed endothelin receptor antagonist, inhibits superoxide anion-induced pain and inflammation in mice
- Curcumin inhibits superoxide anion-induced pain-like behavior and leukocyte recruitment by increasing Nrf2 expression and reducing NF-kB activation
- 15d-PGJ2-loaded nanocapsules ameliorate experimental gout arthritis by reducing pain and inflammation in a PPAR-gamma-sensitive manner in mice
- Probucol attenuates lipopolysaccharide-induced leukocyte recruitment and inflammatory hyperalgesia: effect on NF-кB activation and cytokine production
- Diosmin reduces chronic constriction injury-induced neuropathic pain in mice
- The PI3Kγ/AKT signaling pathway mediates peripheral antinociceptive action of dipyrone
- Targeting IL-33/ST2 signaling: regulation of immune function and analgesia
- Probucol attenuates overt pain-like behavior and carrageenan-induced inflammatory hyperalgesia and leukocyte recruitment by inhibiting NF-кB activation and cytokine production without antioxidant effects
- Pericytes modulate myelination in the central nervous system
Informações sobre o DOI: 10.1016/j.cbi.2015.05.007 (Fonte: oaDOI API)
Como citar
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas