Effects of high fat diets on rodent liver bioenergetics and oxidative imbalance (2016)
- Authors:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- DOI: 10.1016/j.redox.2016.01.009
- Subjects: DOENÇAS METABÓLICAS; MITOCÔNDRIAS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Redox Biology
- ISSN: 2213-2317
- Volume/Número/Paginação/Ano: v. 8, p. 216-225, 2016
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by-nc-nd
-
ABNT
KAKIMOTO, Pâmela A e KOWALTOWSKI, Alicia Juliana. Effects of high fat diets on rodent liver bioenergetics and oxidative imbalance. Redox Biology, v. 8, p. 216-225, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.redox.2016.01.009. Acesso em: 29 mar. 2024. -
APA
Kakimoto, P. A., & Kowaltowski, A. J. (2016). Effects of high fat diets on rodent liver bioenergetics and oxidative imbalance. Redox Biology, 8, 216-225. doi:10.1016/j.redox.2016.01.009 -
NLM
Kakimoto PA, Kowaltowski AJ. Effects of high fat diets on rodent liver bioenergetics and oxidative imbalance [Internet]. Redox Biology. 2016 ; 8 216-225.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1016/j.redox.2016.01.009 -
Vancouver
Kakimoto PA, Kowaltowski AJ. Effects of high fat diets on rodent liver bioenergetics and oxidative imbalance [Internet]. Redox Biology. 2016 ; 8 216-225.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1016/j.redox.2016.01.009 - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
Informações sobre o DOI: 10.1016/j.redox.2016.01.009 (Fonte: oaDOI API)
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