Effects of caloric restriction, dietary restriction and every other day feeding on energy metabolism and redox state (2010)
- Autores:
- Autores USP: COLEPICOLO NETO, PIO - IQ ; KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Assuntos: MICRONUTRIENTES; METABOLISMO ENERGÉTICO
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Biochimica et Biophysica Acta - Bioenergetics
- ISSN: 0005-2728
- Volume/Número/Paginação/Ano: v. 1797, suppl., p. 58 res. 5P.4, 2010
- Nome do evento: European Bioenergetics Conference
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ABNT
CERQUEIRA, Fernanda Menezes et al. Effects of caloric restriction, dietary restriction and every other day feeding on energy metabolism and redox state. Biochimica et Biophysica Acta - Bioenergetics. Amsterdam: Instituto de Química, Universidade de São Paulo. . Acesso em: 24 abr. 2024. , 2010 -
APA
Cerqueira, F. M., Cunha, F. M., Silva, C. C. C. da, Carrião, C., Lahos, R., Colepicolo, P., & Kowaltowski, A. J. (2010). Effects of caloric restriction, dietary restriction and every other day feeding on energy metabolism and redox state. Biochimica et Biophysica Acta - Bioenergetics. Amsterdam: Instituto de Química, Universidade de São Paulo. -
NLM
Cerqueira FM, Cunha FM, Silva CCC da, Carrião C, Lahos R, Colepicolo P, Kowaltowski AJ. Effects of caloric restriction, dietary restriction and every other day feeding on energy metabolism and redox state. Biochimica et Biophysica Acta - Bioenergetics. 2010 ; 1797 58 res. 5P.4.[citado 2024 abr. 24 ] -
Vancouver
Cerqueira FM, Cunha FM, Silva CCC da, Carrião C, Lahos R, Colepicolo P, Kowaltowski AJ. Effects of caloric restriction, dietary restriction and every other day feeding on energy metabolism and redox state. Biochimica et Biophysica Acta - Bioenergetics. 2010 ; 1797 58 res. 5P.4.[citado 2024 abr. 24 ] - Intermittent fasting, but not caloric restriction, leads to redox imbalance, insulin receptor modification and impaired glucose metabolism
- Redox state, insulin sensitivity and aging
- Long-term intermittent feeding, but not caloric restriction, leads to redox imbalance, insulin receptor nitration, and glucose intolerance
- Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
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