G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction (2014)
- Authors:
- USP affiliated authors: CARNEIRO, FERNANDO SILVA - FMRP ; PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - FMRP ; MANZATO, CARLA PAVAN - FMRP
- Unidade: FMRP
- Subjects: ENDOTÉLIO VASCULAR; DIABETES MELLITUS
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2014
- Source:
- Título do periódico: Final Program
- Conference titles: High Blood Pressure Research - Scientific Sessions
-
ABNT
FERREIRA, Nathanne et al. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. 2014, Anais.. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2014. . Acesso em: 24 abr. 2024. -
APA
Ferreira, N., Cau, S. B., Manzato, C. P., Silva, M. A., Carneiro, F. S., & Tostes, R. de C. A. (2014). G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. In Final Program. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Ferreira N, Cau SB, Manzato CP, Silva MA, Carneiro FS, Tostes R de CA. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. Final Program. 2014 ;[citado 2024 abr. 24 ] -
Vancouver
Ferreira N, Cau SB, Manzato CP, Silva MA, Carneiro FS, Tostes R de CA. G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction. Final Program. 2014 ;[citado 2024 abr. 24 ] - Diabetes impairs the vascular effects of aldosterone mediated by G protein-coupled estrogen receptor activation
- Mesenteric arteries from stroke-prone spontaneously hypertensive rats exhibit an increase in nitric-oxide-dependent vasorelaxation
- Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery
- O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway
- Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms
- Angiotensin (1-7) inhibits Ang II-mediated ERK1/2 activation by stimulating MKP-1 activation in vascular smooth muscle cells
- Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats
- Bonus effects of antidiabetic drugs: possible beneficial effects on endothelial dysfunction, vascular inflammation and atherosclerosis
- NACHT, LRR and PYD domains-containing protein 3(NLRP3) crucial role of the cavernous tissue tone modulation in mice
- O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas