Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation (2014)
- Authors:
- USP affiliated authors: GARCIA, CRISTIANA BERNADELLI - FCFRP ; CURTI, CARLOS - FCFRP ; LEOPOLDINO, ANDRÉIA MACHADO - FCFRP
- Unidade: FCFRP
- DOI: 10.1016/j.bbrc.2014.01.175
- Subjects: CARCINOMA DE CÉLULAS ESCAMOSAS; LINHAGEM CELULAR; PROLIFERAÇÃO CELULAR; REGULAÇÃO GÊNICA; NEOPLASIAS DE CABEÇA E PESCOÇO; PROTEÍNAS
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2014
- Source:
- Título do periódico: Biochemical and Biophysical Research Communications
- ISSN: 0006-291X
- Volume/Número/Paginação/Ano: v. 445, n. 1, p. 196\2013202, 2014
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
ALMEIDA, Luciana O. et al. Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation. Biochemical and Biophysical Research Communications, v. 445, n. 1, p. 196\2013202, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2014.01.175. Acesso em: 19 abr. 2024. -
APA
Almeida, L. O., Garcia, C. B., Silva, F. A. M. e, Curti, C., & Leopoldino, A. M. (2014). Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation. Biochemical and Biophysical Research Communications, 445( 1), 196\2013202. doi:10.1016/j.bbrc.2014.01.175 -
NLM
Almeida LO, Garcia CB, Silva FAM e, Curti C, Leopoldino AM. Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation [Internet]. Biochemical and Biophysical Research Communications. 2014 ; 445( 1): 196\2013202.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.bbrc.2014.01.175 -
Vancouver
Almeida LO, Garcia CB, Silva FAM e, Curti C, Leopoldino AM. Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation [Internet]. Biochemical and Biophysical Research Communications. 2014 ; 445( 1): 196\2013202.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.bbrc.2014.01.175 - Set/I2PPA2 plays relevant function in oral tumorigenesis by transcription and signaling regulation
- Autoantibodies against set protein as a potential serum marker in cervical, breast and head and neck cancer patients: detection by ELISA assay
- New insights into heterogenous ribonucleoprotein K expression and accumulation in oral squamous cell carcinoma
- SET/I2PP2A is an essential protein for cell migration in head and neck squamous cell carcinoma
- SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant
- Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation
- SET protects mitochondrial function and induces autophagy in response to redox unbalance
- SET protein regulates expression of glutathione S-transferase pi 1 (GSTP-1) in oral carcinoma
- Different responses of two HNSCC cell lineages to oxidative stress
- Knockdown of SET protein sensitizes HNSCC-HN12 cells to UCN-01
Informações sobre o DOI: 10.1016/j.bbrc.2014.01.175 (Fonte: oaDOI API)
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