Cellular mechanisms involved in the venodilation induced by nitric oxide donors (2010)
- Authors:
- USP affiliated authors: VERCESI, JULIANA APARECIDA - FCFRP ; SILVA, ROBERTO SANTANA DA - FCFRP ; BENDHACK, LUSIANE MARIA - FCFRP
- Unidade: FCFRP
- Subjects: ÓXIDO NÍTRICO; VASODILATAÇÃO; VEIAS
- Language: Inglês
- Imprenta:
- Publisher place: Ribeirão Preto
- Date published: 2010
- Source:
- Título do periódico: Programa
- Conference titles: Congresso Brasileiro de Farmacologia e Terapêutica Experimental
-
ABNT
PAULO, M. et al. Cellular mechanisms involved in the venodilation induced by nitric oxide donors. 2010, Anais.. Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2010. . Acesso em: 19 abr. 2024. -
APA
Paulo, M., Vercesi, J. A., Biazzotto, J. C., Silva, R. S. da, & Bendhack, L. M. (2010). Cellular mechanisms involved in the venodilation induced by nitric oxide donors. In Programa. Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Paulo M, Vercesi JA, Biazzotto JC, Silva RS da, Bendhack LM. Cellular mechanisms involved in the venodilation induced by nitric oxide donors. Programa. 2010 ;[citado 2024 abr. 19 ] -
Vancouver
Paulo M, Vercesi JA, Biazzotto JC, Silva RS da, Bendhack LM. Cellular mechanisms involved in the venodilation induced by nitric oxide donors. Programa. 2010 ;[citado 2024 abr. 19 ] - Vascular smooth muscle relaxation induced by nitric oxide(NO) donor
- Endothelium negatively modulates the vascular relaxation induced by nitric oxide donor, due to uncoupling NO synthase
- The contribution of guanylyl cyclase and potassium channels to the relaxation induced by the NO donors [Ru(terpy)(bdq)NO+]3+ (Terpy) and sodium nitroprusside (SNP) in rat mesenteric resistance arteries
- Nitric oxide (NO) released form NO donors has hypotensive effect and induces rat aorta relaxation due to the activation of K+ channels, guanylyl-cyclase and SERCA
- The nitric oxide (NO) donors [Ru(terpy)(bdq)NO+]3+(TERPY) and sodium nitroprusside (SNP) have different cellular effects in rat basilar arteries relaxation
- Acetylcholine induces relaxation dependent on nitric oxide synthase (NOS) and an alternative pathway during sepsis
- Acetylcholine induces relaxation dependent on nitric oxide synthase and cyclooxigenase during sepsis
- Relaxation induced by nitrosyl compound in rat aorta
- A macrocyclic ruthenium nitrosyl complex as a pontential nitric oxide deliverer mediating vascular smooth muscle relaxation
- The nitric oxide donor cis-[Ru(Cl)(bpy)2(NO)](PF6) induces rat aorta relaxation and Ca2+ reduction under visible light irradiation
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