Inhibition of the inflammasome activation: a survival strategy of Coxiella burnetii ? (2010)
- Authors:
- Autor USP: ZAMBONI, DARIO SIMÕES - FMRP
- Unidade: FMRP
- Subjects: FEBRE Q; BACTÉRIAS GRAM-NEGATIVAS; MACRÓFAGOS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Abstracts
- Conference titles: Annual Meeting of the Society for Leukocyte Biology (SLB)
-
ABNT
CUNHA, Larissa D. e ZAMBONI, Dario Simões. Inhibition of the inflammasome activation: a survival strategy of Coxiella burnetii ? 2010, Anais.. Vancouver: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2010. . Acesso em: 24 abr. 2024. -
APA
Cunha, L. D., & Zamboni, D. S. (2010). Inhibition of the inflammasome activation: a survival strategy of Coxiella burnetii ? In Abstracts. Vancouver: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Cunha LD, Zamboni DS. Inhibition of the inflammasome activation: a survival strategy of Coxiella burnetii ? Abstracts. 2010 ;[citado 2024 abr. 24 ] -
Vancouver
Cunha LD, Zamboni DS. Inhibition of the inflammasome activation: a survival strategy of Coxiella burnetii ? Abstracts. 2010 ;[citado 2024 abr. 24 ] - Role of inflammasomes in diffuse and localized cutaneous Leishmaniasis
- Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to Leishmania major infection
- Role of the transcriptional regulator SP140 in resistance to bacterial infections via repression of type I interferons
- Unique role of the Nlrc4 inflammasome for pore formation and pyroptosis in response to flagellated bacterial pathogens
- Nod-like receptors-dependent innate immune responses to intracellular pathogens
- NALP3: a key player in caspase-1 activation
- NOD1 and NOD2 signaling in infection and inflammation
- Participação dos receptores de reconhecimento padrão NOD1 e NOD2 no recrutamento de neutrófilos para o pulmão em resposta a infecção murina por legionella penumophila
- Critical role of ASC inflammasomes and bacterial type IV secretion system in caspase-1 activation and host innate resistance to Brucella abortus infection
- Inflammasome mediated innate immune responses to Leishmania
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