A novel pathway for inducible nitric oxide synthase (iNOS) activation through inflammasomes (2010)
- Authors:
- Lima, Carina Buzzo de - Universidade de São Paulo (USP)
- Campopiano, Júlia Cortina - Universidade de São Paulo (USP)
- Massis, Liliana Moura
- Lage, Silvia Lucena - Universidade de São Paulo (USP)
- Cassado, Alexandra dos Anjos - Universidade de São Paulo (USP)
- Leme-Souza, Rafael
- Cunha, Larissa Dias da
- Momtchilo, Russo
- Zamboni, Dario Simões
- Amarante-Mendes, João Gustavo Pessini
- Bortoluci, Karina Ramalho
- USP affiliated authors: RUSSO, MOMTCHILO - ICB ; ZAMBONI, DARIO SIMÕES - FMRP ; MENDES, JOAO GUSTAVO PESSINI AMARANTE - ICB
- Unidades: ICB; FMRP
- DOI: 10.1074/jbc.M110.124297
- Subjects: ÓXIDO NÍTRICO; MACRÓFAGOS; SISTEMA IMUNE; IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Biological Chemistry
- ISSN: 0021-9258
- Volume/Número/Paginação/Ano: v. 285, n. 42, p. 32087-32095, 2010
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: cc-by
-
ABNT
LIMA, Carina Buzzo de et al. A novel pathway for inducible nitric oxide synthase (iNOS) activation through inflammasomes. Journal of Biological Chemistry, v. 285, n. 42, p. 32087-32095, 2010Tradução . . Disponível em: https://doi.org/10.1074/jbc.M110.124297. Acesso em: 19 abr. 2024. -
APA
Lima, C. B. de, Campopiano, J. C., Massis, L. M., Lage, S. L., Cassado, A. dos A., Leme-Souza, R., et al. (2010). A novel pathway for inducible nitric oxide synthase (iNOS) activation through inflammasomes. Journal of Biological Chemistry, 285( 42), 32087-32095. doi:10.1074/jbc.M110.124297 -
NLM
Lima CB de, Campopiano JC, Massis LM, Lage SL, Cassado A dos A, Leme-Souza R, Cunha LD da, Momtchilo R, Zamboni DS, Amarante-Mendes JGP, Bortoluci KR. A novel pathway for inducible nitric oxide synthase (iNOS) activation through inflammasomes [Internet]. Journal of Biological Chemistry. 2010 ; 285( 42): 32087-32095.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1074/jbc.M110.124297 -
Vancouver
Lima CB de, Campopiano JC, Massis LM, Lage SL, Cassado A dos A, Leme-Souza R, Cunha LD da, Momtchilo R, Zamboni DS, Amarante-Mendes JGP, Bortoluci KR. A novel pathway for inducible nitric oxide synthase (iNOS) activation through inflammasomes [Internet]. Journal of Biological Chemistry. 2010 ; 285( 42): 32087-32095.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1074/jbc.M110.124297 - A suggestive role of naip5 on inos activation in flagellin-stimulated macrophages
- Regulação do CD95L por PGE2 e seu impacto na morte de linfócitos T.
- Activation of inos trough NAIP5/NLRC4/CASPASE-1 AXIS
- A suggestive role of NAIP5 on INOS activation
- TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3-mediated CD95L upregulation in T cells
- A novel pathway for inducible nitric oxide synthase activation by cytosolic flagellin dependent on caspase-1
- Inhibition of interferon-'gama'-induced nitric oxide production in endotoxin-activated macrophages by cytolethal distending toxin
- Cross-talk between innate and adaptive immunity: TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3- mediated apoptosis of T cells
- PGE2 derivada de macrófagos ou células dendríticas protege linfócitos T da AICD bloqueando a expressão de FasL
- FASL upregulation and T cell death by AICD are prevented by macrophage- and DC-derived PGE2
Informações sobre o DOI: 10.1074/jbc.M110.124297 (Fonte: oaDOI API)
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