LH-induced depolarization of membrane potential in mice Leydig cells can function as a trigger for the subsequent intracellular calcium increase (2008)
- Authors:
- USP affiliated authors: AGUIAR, JOSE FERNANDO - FMRP ; VARANDA, WAMBERTO ANTONIO - FMRP
- Unidade: FMRP
- Subjects: TESTOSTERONA; CÁLCIO; CÉLULAS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Abstracts
- Conference titles: Meeting of the Physiological Society
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ABNT
COSTA, R. R. e AGUIAR, José Fernando e VARANDA, Wamberto Antônio. LH-induced depolarization of membrane potential in mice Leydig cells can function as a trigger for the subsequent intracellular calcium increase. 2008, Anais.. Cambridge: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2008. . Acesso em: 24 abr. 2024. -
APA
Costa, R. R., Aguiar, J. F., & Varanda, W. A. (2008). LH-induced depolarization of membrane potential in mice Leydig cells can function as a trigger for the subsequent intracellular calcium increase. In Abstracts. Cambridge: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Costa RR, Aguiar JF, Varanda WA. LH-induced depolarization of membrane potential in mice Leydig cells can function as a trigger for the subsequent intracellular calcium increase. Abstracts. 2008 ;[citado 2024 abr. 24 ] -
Vancouver
Costa RR, Aguiar JF, Varanda WA. LH-induced depolarization of membrane potential in mice Leydig cells can function as a trigger for the subsequent intracellular calcium increase. Abstracts. 2008 ;[citado 2024 abr. 24 ] - Cross-talk between G-protein-coupled and P2X receptors in mice leydig cells
- PKA mediates intracellular calcium increase induced by LH in mice Leydig cells by modulating Cav.3.2 channels
- LH induced depolarization of membrane potential in mice leydig cells may function as a trigger for the subsequent intracellular calcium increase
- Luteinizing hormone (LH) acts through PKA and PKC to modulate T-type calcium currents and intracellular calcium transients in mice Leydig cells
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- Effects of IgG from patients with idiopathic dilated cardiomyopathy on L-type calcium currents in rabbit cardiomyocytes
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