Effect of a high fat diet on mitochondrial ATP sensitive potassium channels (mitoKATP), a mild uncoupling pathway (2008)
- Autores:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Assuntos: MITOCÔNDRIAS; POTÁSSIO; DOENÇAS METABÓLICAS
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Abstracts
- Nome do evento: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq)
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ABNT
CARDOSO, Ariel Rodrigues e KOWALTOWSKI, Alicia Juliana. Effect of a high fat diet on mitochondrial ATP sensitive potassium channels (mitoKATP), a mild uncoupling pathway. 2008, Anais.. São Paulo: SBBq, 2008. . Acesso em: 19 abr. 2024. -
APA
Cardoso, A. R., & Kowaltowski, A. J. (2008). Effect of a high fat diet on mitochondrial ATP sensitive potassium channels (mitoKATP), a mild uncoupling pathway. In Abstracts. São Paulo: SBBq. -
NLM
Cardoso AR, Kowaltowski AJ. Effect of a high fat diet on mitochondrial ATP sensitive potassium channels (mitoKATP), a mild uncoupling pathway. Abstracts. 2008 ;[citado 2024 abr. 19 ] -
Vancouver
Cardoso AR, Kowaltowski AJ. Effect of a high fat diet on mitochondrial ATP sensitive potassium channels (mitoKATP), a mild uncoupling pathway. Abstracts. 2008 ;[citado 2024 abr. 19 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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