Mitochondrial ATP-sensitive K+ channels ara activated by reactive oxygen and nitrogen species (2008)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; MITOCÔNDRIAS; ISQUEMIA MIOCÁRDICA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Abstracts
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq)
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ABNT
QUELICONI, Bruno Barros e FACUNDO, Heberty Di Tarso Fernandes e KOWALTOWSKI, Alicia Juliana. Mitochondrial ATP-sensitive K+ channels ara activated by reactive oxygen and nitrogen species. 2008, Anais.. São Paulo: SBBq, 2008. . Acesso em: 18 abr. 2024. -
APA
Queliconi, B. B., Facundo, H. D. T. F., & Kowaltowski, A. J. (2008). Mitochondrial ATP-sensitive K+ channels ara activated by reactive oxygen and nitrogen species. In Abstracts. São Paulo: SBBq. -
NLM
Queliconi BB, Facundo HDTF, Kowaltowski AJ. Mitochondrial ATP-sensitive K+ channels ara activated by reactive oxygen and nitrogen species. Abstracts. 2008 ;[citado 2024 abr. 18 ] -
Vancouver
Queliconi BB, Facundo HDTF, Kowaltowski AJ. Mitochondrial ATP-sensitive K+ channels ara activated by reactive oxygen and nitrogen species. Abstracts. 2008 ;[citado 2024 abr. 18 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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