Neutrophil haptotaxis induced by mouse MNCF: interactions with extracellular matrix glycoproteins probably contribute to overcoming the anti-inflammatory action of dexamethasone (2007)
- Autores:
- Autores USP: BARUFFI, MARCELO DIAS - FCFRP ; BARREIRA, MARIA CRISTINA ROQUE ANTUNES - FMRP
- Unidades: FCFRP; FMRP
- DOI: 10.1007/s00011-007-6159-6
- Assuntos: BIOLOGIA CELULAR; ANTI-INFLAMATÓRIOS
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Inflamation Research
- ISSN: 1023-3830
- Volume/Número/Paginação/Ano: v. 56, p. 368-376, 2007
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
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ABNT
TOLEDO, Karina Alves de et al. Neutrophil haptotaxis induced by mouse MNCF: interactions with extracellular matrix glycoproteins probably contribute to overcoming the anti-inflammatory action of dexamethasone. Inflamation Research, v. 56, p. 368-376, 2007Tradução . . Disponível em: https://doi.org/10.1007/s00011-007-6159-6. Acesso em: 19 set. 2024. -
APA
Toledo, K. A. de, Bernardes, E. S., Dias-Baruffi, M., & Roque-Barreira, M. C. (2007). Neutrophil haptotaxis induced by mouse MNCF: interactions with extracellular matrix glycoproteins probably contribute to overcoming the anti-inflammatory action of dexamethasone. Inflamation Research, 56, 368-376. doi:10.1007/s00011-007-6159-6 -
NLM
Toledo KA de, Bernardes ES, Dias-Baruffi M, Roque-Barreira MC. Neutrophil haptotaxis induced by mouse MNCF: interactions with extracellular matrix glycoproteins probably contribute to overcoming the anti-inflammatory action of dexamethasone [Internet]. Inflamation Research. 2007 ; 56 368-376.[citado 2024 set. 19 ] Available from: https://doi.org/10.1007/s00011-007-6159-6 -
Vancouver
Toledo KA de, Bernardes ES, Dias-Baruffi M, Roque-Barreira MC. Neutrophil haptotaxis induced by mouse MNCF: interactions with extracellular matrix glycoproteins probably contribute to overcoming the anti-inflammatory action of dexamethasone [Internet]. Inflamation Research. 2007 ; 56 368-376.[citado 2024 set. 19 ] Available from: https://doi.org/10.1007/s00011-007-6159-6 - Galectin-3 participantes in the inital recruitment of neutrophils during acute inflammatory response by means of a haptotatic mechanism
- Terminal 'alfa'-galactosyl glycans are recognized by MNCF, a neutrophil attractant lectin released by activated macrophages
- Heparin potentiates neutrophil migration induced in vivo by il-8
- Heparina potencializa a quimiotracao de neutrofilos induzida in vivo por il-8
- Galectin-1-matured myeloid dendritic cells produce TNF-'alfa'
- Novos estudos sobre o fator quimiotático para neutrófilos derivados de macrófagos (MNCF)
- Macrófagos murinos produzem um fator similar ao MNCF de rato
- Heparin potentiates neutrophil migration induced in vivo by il-8
- Heparin potentiates in vivo neutrophil migration induced by IL-8
- Murine macrophages produces a neutrophil chemotactic factor similar to rat MNCF
Informações sobre o DOI: 10.1007/s00011-007-6159-6 (Fonte: oaDOI API)
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